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10.1016/j.cellimm.2020.104114

http://scihub22266oqcxt.onion/10.1016/j.cellimm.2020.104114
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suck abstract from ncbi


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pmid32361409      Cell+Immunol 2020 ; 353 (ä): 104114
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  • Protective immunity after COVID-19 has been questioned: What can we do without SARS-CoV-2-IgG detection? #MMPMID32361409
  • Melgaco JG; Azamor T; Ano Bom APD
  • Cell Immunol 2020[Jul]; 353 (ä): 104114 PMID32361409show ga
  • Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) induces a severe acute respiratory syndrome that is called COVID-19. Clinical manifestations of COVID-19 include diarrhea, pneumonia, lymphopenia, exhausted lymphocytes, and pro-inflammatory cytokine production. Immunology is part of the process of clinical evolution, but there are some questions around immunity-based protection: (1) why some infected people have only mild symptoms of the disease or are asymptomatic; (2) why delayed and weak antibody responses are associated with severe outcomes; and (3) why positivity in molecular tests does not represent protective antibody IgG. Perhaps T cell responses may be the key to solving those questions. SARS-CoV-2-specific memory T cells persist in peripheral blood and may be capable of providing effective information about protective immunity. The T cells studies can be helpful in elucidating the pathways for development of vaccines, therapies, and diagnostics for COVID-19 and for filling these immunology knowledge gaps.
  • |Antibodies, Viral/*immunology[MESH]
  • |Antibody Formation[MESH]
  • |Betacoronavirus/*physiology[MESH]
  • |COVID-19[MESH]
  • |COVID-19 Testing[MESH]
  • |Clinical Laboratory Techniques[MESH]
  • |Coronavirus Infections/diagnosis/*immunology/transmission[MESH]
  • |Humans[MESH]
  • |Immunoglobulin G/*immunology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*immunology/transmission[MESH]
  • |SARS-CoV-2[MESH]


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