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10.1111/eci.13259 http://scihub22266oqcxt.onion/10.1111/eci.13259 32353898!7267318!32353898     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Eur+J+Clin+Invest 2020 ; 50 (7): e13259 Nephropedia Template TP {{tp|p=32353898|t=2020. Histopathology and genetic susceptibility in COVID-19 pneumonia |pdf=|usr=}}{{32353898}} gab.com Text Histopathology and genetic susceptibility in COVID-19 pneumonia JO: Eur J Clin Invest http://www.kidney.de/pget.php?&tw=1&pmid=32353898 #FOAvip BACKGROUND: The clinical features of COVID-19 pneumonia range from a mild illness to patients with a very severe illness with acute hypoxemic respiratory failure requiring ventilation and Intensive Care Unit admission. AIMS: To provide a brief overview of the existing evidence for such differences in host response and outcome, and generate hypotheses for divergent patterns and avenues for future research, by highlighting similarities and differences in histopathological appearance between COVID-19 and influenza as well as previous coronavirus outbreaks, and by discussing predisposition through genetics and underlying disease. MATERIALS AND METHOD: We assessed the available early literature for histopathological patterns of COVID-19 pneumonia and underlying risk factors. RESULT: The histopathological spectrum of COVID-19 pneumonia includes variable patterns of epithelial damage, vascular complications, fibrosis and inflammation. Risk factors for a fatal disease include older age, respiratory disease, diabetes mellitus, obesity and hypertension. DISCUSSION: While some risk factors and their potential role in COVID-19 pneumonia are increasingly recognized, little is known about the mechanisms behind episodes of sudden deterioration or the infrequent idiosyncratic clinical demise in otherwise healthy and young subjects. CONCLUSION: The answer to many of the remaining questions regarding COVID-19 pneumonia pathogenesis may in time be provided by genotyping as well careful clinical, serological, radiological and histopathological phenotyping. Twit Text FOAVip Histopathology and genetic susceptibility in COVID-19 pneumonia ????Eur J Clin Invest http://www.kidney.de/pget.php?&tw=1&pmid=32353898 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32353898 #FOAvip English Wikipedia <ref>{{Cite pmid|32353898}}</ref> Histopathology and genetic susceptibility in COVID-19 pneumonia #MMPMID32353898 von der Thusen J; van der Eerden M Eur J Clin Invest 2020[Jul]; 50 (7): e13259 PMID32353898show ga BACKGROUND: The clinical features of COVID-19 pneumonia range from a mild illness to patients with a very severe illness with acute hypoxemic respiratory failure requiring ventilation and Intensive Care Unit admission. AIMS: To provide a brief overview of the existing evidence for such differences in host response and outcome, and generate hypotheses for divergent patterns and avenues for future research, by highlighting similarities and differences in histopathological appearance between COVID-19 and influenza as well as previous coronavirus outbreaks, and by discussing predisposition through genetics and underlying disease. MATERIALS AND METHOD: We assessed the available early literature for histopathological patterns of COVID-19 pneumonia and underlying risk factors. RESULT: The histopathological spectrum of COVID-19 pneumonia includes variable patterns of epithelial damage, vascular complications, fibrosis and inflammation. Risk factors for a fatal disease include older age, respiratory disease, diabetes mellitus, obesity and hypertension. DISCUSSION: While some risk factors and their potential role in COVID-19 pneumonia are increasingly recognized, little is known about the mechanisms behind episodes of sudden deterioration or the infrequent idiosyncratic clinical demise in otherwise healthy and young subjects. CONCLUSION: The answer to many of the remaining questions regarding COVID-19 pneumonia pathogenesis may in time be provided by genotyping as well careful clinical, serological, radiological and histopathological phenotyping. |Age Factors[MESH] |Angiotensin-Converting Enzyme 2[MESH] |Betacoronavirus[MESH] |COVID-19[MESH] |Coronavirus Infections/epidemiology/genetics/immunology/*pathology[MESH] |Cytokine Release Syndrome/immunology[MESH] |Diabetes Mellitus/epidemiology[MESH] |Edema/*pathology[MESH] |Fibrosis[MESH] |Genetic Predisposition to Disease[MESH] |HLA Antigens/genetics[MESH] |Humans[MESH] |Hypertension/epidemiology[MESH] |Inflammation/immunology/*pathology[MESH] |Influenza, Human/pathology[MESH] |Obesity/epidemiology[MESH] |Pandemics[MESH] |Peptidyl-Dipeptidase A/genetics[MESH] |Pneumonia, Viral/epidemiology/genetics/immunology/*pathology[MESH] |Polymorphism, Genetic[MESH] |Respiratory Mucosa/immunology/*pathology[MESH] |Respiratory System/pathology[MESH] |Risk Factors[MESH] |SARS-CoV-2[MESH] |Serine Endopeptidases/genetics[MESH] |Severe Acute Respiratory Syndrome/pathology[MESH]Histopathology and genetic susceptibility in COVID-19 pneumonia (epmc).pdf DeepDyve Pubget Overpricing