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10.1183/13993003.01028-2020 http://scihub22266oqcxt.onion/10.1183/13993003.01028-2020 32341111!7236837!32341111     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Eur+Respir+J 2020 ; 55 (5): ä Nephropedia Template TP {{tp|p=32341111|t=2020. Clinical phenotypes of SARS-CoV-2: implications for clinicians and researchers |pdf=|usr=}}{{32341111}} gab.com Text Clinical phenotypes of SARS-CoV-2: implications for clinicians and researchers JO: Eur Respir J http://www.kidney.de/pget.php?&tw=1&pmid=32341111 #FOAvip Patients with COVID-19 present a broad spectrum of clinical presentation. Whereas hypoxaemia is the marker of severity, different strategies of management should be customised to five specific individual phenotypes. Many intubated patients present with phenotype 4, characterised by pulmonary hypoxic vasoconstriction, being associated with severe hypoxaemia with "normal" (>40 mL.cmH(2)O(-1)) lung compliance and likely representing pulmonary microvascular thrombosis. Phenotype 5 is often associated with high plasma procalcitonin and has low pulmonary compliance, Which is a result of co-infection or acute lung injury after noninvasive ventilation. Identifying these clinical phenotypes and applying a personalised approach would benefit the optimisation of therapies and improve outcomes. Twit Text FOAVip Clinical phenotypes of SARS-CoV-2: implications for clinicians and researchers ????Eur Respir J http://www.kidney.de/pget.php?&tw=1&pmid=32341111 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32341111 #FOAvip English Wikipedia <ref>{{Cite pmid|32341111}}</ref> Clinical phenotypes of SARS-CoV-2: implications for clinicians and researchers #MMPMID32341111 Rello J; Storti E; Belliato M; Serrano R Eur Respir J 2020[May]; 55 (5): ä PMID32341111show ga Patients with COVID-19 present a broad spectrum of clinical presentation. Whereas hypoxaemia is the marker of severity, different strategies of management should be customised to five specific individual phenotypes. Many intubated patients present with phenotype 4, characterised by pulmonary hypoxic vasoconstriction, being associated with severe hypoxaemia with "normal" (>40 mL.cmH(2)O(-1)) lung compliance and likely representing pulmonary microvascular thrombosis. Phenotype 5 is often associated with high plasma procalcitonin and has low pulmonary compliance, Which is a result of co-infection or acute lung injury after noninvasive ventilation. Identifying these clinical phenotypes and applying a personalised approach would benefit the optimisation of therapies and improve outcomes. |*Phenotype[MESH] |Acute Lung Injury/*physiopathology/therapy/virology[MESH] |Betacoronavirus/*genetics[MESH] |Biomarkers/blood[MESH] |Biomedical Research[MESH] |COVID-19[MESH] |Coronavirus Infections/*genetics/therapy[MESH] |Disease Management[MESH] |Female[MESH] |Humans[MESH] |Hypoxia/diagnosis/etiology[MESH] |Lung Compliance/genetics[MESH] |Male[MESH] |Pandemics[MESH] |Pneumonia, Viral/*genetics/therapy[MESH] |Procalcitonin/metabolism[MESH] |SARS-CoV-2[MESH]Clinical phenotypes of SARS-CoV-2: implications for clinicians and res(epmc).pdf DeepDyve Pubget Overpricing