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10.1016/j.resp.2020.103443 http://scihub22266oqcxt.onion/10.1016/j.resp.2020.103443 32333993!7175867!32333993     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32333993&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Respir+Physiol+Neurobiol 2020 ; 277 (?): 103443 Nephropedia Template TP {{tp|p=32333993|t=2020. Does the pathogenesis of SARS-CoV-2 virus decrease at high-altitude?|pdf=|usr=}}{{32333993}} gab.com Text Does the pathogenesis of SARS-CoV-2 virus decrease at high-altitude JO: Respir Physiol Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=32333993 #FOAvip In the present study we analyze the epidemiological data of COVID-19 of Tibet and high-altitude regions of Bolivia and Ecuador, and compare to lowland data, to test the hypothesis that high-altitude inhabitants (+2,500 m above sea-level) are less susceptible to develop severe adverse effects in acute SARS-CoV-2 virus infection. Analysis of available epidemiological data suggest that physiological acclimatization/adaptation that counterbalance the hypoxic environment in high-altitude may protect from severe impact of acute SARS-CoV-2 virus infection. Potential underlying mechanisms such as: (i) a compromised half-live of the virus caused by the high-altitude environment, and (ii) a hypoxia mediated down regulation of angiotensin-converting enzyme 2 (ACE2), which is the main binding target of SARS-CoV-2 virus in the pulmonary epithelium are discussed. Twit Text FOAVip Does the pathogenesis of SARS-CoV-2 virus decrease at high-altitude ????Respir Physiol Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=32333993 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32333993 #FOAvip English Wikipedia <ref>{{Cite pmid|32333993}}</ref> Does the pathogenesis of SARS-CoV-2 virus decrease at high-altitude? #MMPMID32333993 Arias-Reyes C; Zubieta-DeUrioste N; Poma-Machicao L; Aliaga-Raduan F; Carvajal-Rodriguez F; Dutschmann M; Schneider-Gasser EM; Zubieta-Calleja G; Soliz J Respir Physiol Neurobiol 2020[Jun]; 277 (?): 103443 PMID32333993show ga In the present study we analyze the epidemiological data of COVID-19 of Tibet and high-altitude regions of Bolivia and Ecuador, and compare to lowland data, to test the hypothesis that high-altitude inhabitants (+2,500 m above sea-level) are less susceptible to develop severe adverse effects in acute SARS-CoV-2 virus infection. Analysis of available epidemiological data suggest that physiological acclimatization/adaptation that counterbalance the hypoxic environment in high-altitude may protect from severe impact of acute SARS-CoV-2 virus infection. Potential underlying mechanisms such as: (i) a compromised half-live of the virus caused by the high-altitude environment, and (ii) a hypoxia mediated down regulation of angiotensin-converting enzyme 2 (ACE2), which is the main binding target of SARS-CoV-2 virus in the pulmonary epithelium are discussed. |Altitude[MESH] |Betacoronavirus/pathogenicity/*physiology[MESH] |Bolivia/epidemiology[MESH] |COVID-19[MESH] |Coronavirus Infections/*epidemiology/*virology[MESH] |Disease Susceptibility[MESH] |Ecuador/epidemiology[MESH] |Humans[MESH] |Oxygen[MESH] |Pandemics[MESH] |Pneumonia, Viral/*epidemiology/*virology[MESH] |SARS-CoV-2[MESH] |Tibet/epidemiology[MESH]Does the pathogenesis of SARS-CoV-2 virus decrease at high-altitude?(epmc).pdf DeepDyve Pubget Overpricing