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suck abstract from ncbi


10.1016/j.clim.2020.108427

http://scihub22266oqcxt.onion/10.1016/j.clim.2020.108427
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32325252!7169933!32325252
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suck abstract from ncbi

pmid32325252      Clin+Immunol 2020 ; 215 (ä): 108427
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  • COVID-19 pathophysiology: A review #MMPMID32325252
  • Yuki K; Fujiogi M; Koutsogiannaki S
  • Clin Immunol 2020[Jun]; 215 (ä): 108427 PMID32325252show ga
  • In December 2019, a novel coronavirus, now named as SARS-CoV-2, caused a series of acute atypical respiratory diseases in Wuhan, Hubei Province, China. The disease caused by this virus was termed COVID-19. The virus is transmittable between humans and has caused pandemic worldwide. The number of death tolls continues to rise and a large number of countries have been forced to do social distancing and lockdown. Lack of targeted therapy continues to be a problem. Epidemiological studies showed that elder patients were more susceptible to severe diseases, while children tend to have milder symptoms. Here we reviewed the current knowledge about this disease and considered the potential explanation of the different symptomatology between children and adults.
  • |*Pandemics[MESH]
  • |Age Factors[MESH]
  • |Aged[MESH]
  • |Angiotensin-Converting Enzyme 2[MESH]
  • |Animals[MESH]
  • |Betacoronavirus/immunology/*pathogenicity[MESH]
  • |COVID-19[MESH]
  • |Child[MESH]
  • |Coronavirus Infections/*epidemiology/immunology/*physiopathology/virology[MESH]
  • |Cytokines/genetics/immunology[MESH]
  • |Disease Progression[MESH]
  • |Disease Susceptibility[MESH]
  • |Gene Expression Regulation[MESH]
  • |Host-Pathogen Interactions/genetics/immunology[MESH]
  • |Humans[MESH]
  • |Mice[MESH]
  • |Peptidyl-Dipeptidase A/*genetics/immunology[MESH]
  • |Pneumonia, Viral/*epidemiology/immunology/*physiopathology/virology[MESH]
  • |SARS-CoV-2[MESH]
  • |Signal Transduction[MESH]
  • |Spike Glycoprotein, Coronavirus/*genetics/immunology[MESH]


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