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10.1002/dvdy.182 http://scihub22266oqcxt.onion/10.1002/dvdy.182 32315468!ä!32315468 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Dev+Dyn 2020 ; 249 (8): 912-923 Nephropedia Template TP {{tp|p=32315468|t=2020. TRPM6 and TRPM7: Novel players in cell intercalation during vertebrate embryonic development |pdf=|usr=}}{{32315468}} gab.com Text TRPM6 and TRPM7: Novel players in cell intercalation during vertebrate embryonic development JO: Dev Dyn http://www.kidney.de/pget.php?&tw=1&pmid=32315468 #FOAvip A common theme in organogenesis is how the final structure of organs emerge from epithelial tube structures, with the formation of the neural tube being one of the best examples. Two types of cell movements co-occur during neural tube closure involving the migration of cells toward the midline of the embryo (mediolateral intercalation or convergent extension) as well as the deep movement of cells from inside the embryo to the outside of the lateral side of the neural plate (radial intercalation). Failure of either type of cell movement will prevent neural tube closure, which can produce a range of neural tube defects (NTDs), a common congenital disease in humans. Numerous studies have identified signaling pathways that regulate mediolateral intercalation during neural tube closure. Less understood are the pathways that govern radial intercalation. Using the Xenopus laevis system, our group reported the identification of transient receptor potential (TRP) channels, TRPM6 and TRPM7, and the Mg(2+) ion they conduct, as novel and key factors regulating both mediolateral and radial intercalation during neural tube closure. Here we broadly discuss tubulogenesis and cell intercalation from the perspective of neural tube closure and the respective roles of TRPM7 and TRPM6 in this critical embryonic process. Twit Text FOAVip TRPM6 and TRPM7: Novel players in cell intercalation during vertebrate embryonic development ????Dev Dyn http://www.kidney.de/pget.php?&tw=1&pmid=32315468 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32315468 #FOAvip English Wikipedia <ref>{{Cite pmid|32315468}}</ref> TRPM6 and TRPM7: Novel players in cell intercalation during vertebrate embryonic development #MMPMID32315468 Runnels LW; Komiya Y Dev Dyn 2020[Aug]; 249 (8): 912-923 PMID32315468show ga A common theme in organogenesis is how the final structure of organs emerge from epithelial tube structures, with the formation of the neural tube being one of the best examples. Two types of cell movements co-occur during neural tube closure involving the migration of cells toward the midline of the embryo (mediolateral intercalation or convergent extension) as well as the deep movement of cells from inside the embryo to the outside of the lateral side of the neural plate (radial intercalation). Failure of either type of cell movement will prevent neural tube closure, which can produce a range of neural tube defects (NTDs), a common congenital disease in humans. Numerous studies have identified signaling pathways that regulate mediolateral intercalation during neural tube closure. Less understood are the pathways that govern radial intercalation. Using the Xenopus laevis system, our group reported the identification of transient receptor potential (TRP) channels, TRPM6 and TRPM7, and the Mg(2+) ion they conduct, as novel and key factors regulating both mediolateral and radial intercalation during neural tube closure. Here we broadly discuss tubulogenesis and cell intercalation from the perspective of neural tube closure and the respective roles of TRPM7 and TRPM6 in this critical embryonic process. |*Gene Expression Regulation, Developmental[MESH] |3T3 Cells[MESH] |Animals[MESH] |Cell Movement[MESH] |Embryonic Development[MESH] |Humans[MESH] |Ions[MESH] |Magnesium/*chemistry/metabolism[MESH] |Mice[MESH] |Neural Plate/metabolism[MESH] |Neural Tube Defects/genetics/metabolism[MESH] |Neural Tube/metabolism[MESH] |Neurulation[MESH] |Protein Domains[MESH] |Protein Serine-Threonine Kinases/genetics/*physiology[MESH] |Signal Transduction[MESH] |TRPM Cation Channels/genetics/metabolism/*physiology[MESH] |Xenopus Proteins/genetics/*physiology[MESH] |Xenopus laevis/*metabolism[MESH]TRPM6 and TRPM7: Novel players in cell intercalation during vertebrate(epmc).pdf DeepDyve Pubget Overpricing