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10.1084/jem.20200652

http://scihub22266oqcxt.onion/10.1084/jem.20200652
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32302401!7161085!32302401
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suck abstract from ncbi


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pmid32302401      J+Exp+Med 2020 ; 217 (6): ä
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  • Targeting potential drivers of COVID-19: Neutrophil extracellular traps #MMPMID32302401
  • Barnes BJ; Adrover JM; Baxter-Stoltzfus A; Borczuk A; Cools-Lartigue J; Crawford JM; Dassler-Plenker J; Guerci P; Huynh C; Knight JS; Loda M; Looney MR; McAllister F; Rayes R; Renaud S; Rousseau S; Salvatore S; Schwartz RE; Spicer JD; Yost CC; Weber A; Zuo Y; Egeblad M
  • J Exp Med 2020[Jun]; 217 (6): ä PMID32302401show ga
  • Coronavirus disease 2019 (COVID-19) is a novel, viral-induced respiratory disease that in approximately 10-15% of patients progresses to acute respiratory distress syndrome (ARDS) triggered by a cytokine storm. In this Perspective, autopsy results and literature are presented supporting the hypothesis that a little known yet powerful function of neutrophils-the ability to form neutrophil extracellular traps (NETs)-may contribute to organ damage and mortality in COVID-19. We show lung infiltration of neutrophils in an autopsy specimen from a patient who succumbed to COVID-19. We discuss prior reports linking aberrant NET formation to pulmonary diseases, thrombosis, mucous secretions in the airways, and cytokine production. If our hypothesis is correct, targeting NETs directly and/or indirectly with existing drugs may reduce the clinical severity of COVID-19.
  • |*Betacoronavirus[MESH]
  • |*Extracellular Traps[MESH]
  • |*Lung Diseases/etiology/metabolism/pathology[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/complications/*pathology[MESH]
  • |Cytokines/metabolism[MESH]
  • |Humans[MESH]
  • |Neutrophils/*pathology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/complications/*pathology[MESH]


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