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10.1084/jem.20200652 http://scihub22266oqcxt.onion/10.1084/jem.20200652 32302401!7161085!32302401     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\32302401.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Exp+Med 2020 ; 217 (6): ä Nephropedia Template TP {{tp|p=32302401|t=2020. Targeting potential drivers of COVID-19: Neutrophil extracellular traps |pdf=|usr=}}{{32302401}} gab.com Text Targeting potential drivers of COVID-19: Neutrophil extracellular traps JO: J Exp Med http://www.kidney.de/pget.php?&tw=1&pmid=32302401 #FOAvip Coronavirus disease 2019 (COVID-19) is a novel, viral-induced respiratory disease that in approximately 10-15% of patients progresses to acute respiratory distress syndrome (ARDS) triggered by a cytokine storm. In this Perspective, autopsy results and literature are presented supporting the hypothesis that a little known yet powerful function of neutrophils-the ability to form neutrophil extracellular traps (NETs)-may contribute to organ damage and mortality in COVID-19. We show lung infiltration of neutrophils in an autopsy specimen from a patient who succumbed to COVID-19. We discuss prior reports linking aberrant NET formation to pulmonary diseases, thrombosis, mucous secretions in the airways, and cytokine production. If our hypothesis is correct, targeting NETs directly and/or indirectly with existing drugs may reduce the clinical severity of COVID-19. Twit Text FOAVip Targeting potential drivers of COVID-19: Neutrophil extracellular traps ????J Exp Med http://www.kidney.de/pget.php?&tw=1&pmid=32302401 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32302401 #FOAvip English Wikipedia <ref>{{Cite pmid|32302401}}</ref> Targeting potential drivers of COVID-19: Neutrophil extracellular traps #MMPMID32302401 Barnes BJ; Adrover JM; Baxter-Stoltzfus A; Borczuk A; Cools-Lartigue J; Crawford JM; Dassler-Plenker J; Guerci P; Huynh C; Knight JS; Loda M; Looney MR; McAllister F; Rayes R; Renaud S; Rousseau S; Salvatore S; Schwartz RE; Spicer JD; Yost CC; Weber A; Zuo Y; Egeblad M J Exp Med 2020[Jun]; 217 (6): ä PMID32302401show ga Coronavirus disease 2019 (COVID-19) is a novel, viral-induced respiratory disease that in approximately 10-15% of patients progresses to acute respiratory distress syndrome (ARDS) triggered by a cytokine storm. In this Perspective, autopsy results and literature are presented supporting the hypothesis that a little known yet powerful function of neutrophils-the ability to form neutrophil extracellular traps (NETs)-may contribute to organ damage and mortality in COVID-19. We show lung infiltration of neutrophils in an autopsy specimen from a patient who succumbed to COVID-19. We discuss prior reports linking aberrant NET formation to pulmonary diseases, thrombosis, mucous secretions in the airways, and cytokine production. If our hypothesis is correct, targeting NETs directly and/or indirectly with existing drugs may reduce the clinical severity of COVID-19. |*Betacoronavirus[MESH] |*Extracellular Traps[MESH] |*Lung Diseases/etiology/metabolism/pathology[MESH] |COVID-19[MESH] |Coronavirus Infections/complications/*pathology[MESH] |Cytokines/metabolism[MESH] |Humans[MESH] |Neutrophils/*pathology[MESH] |Pandemics[MESH] |Pneumonia, Viral/complications/*pathology[MESH]Targeting potential drivers of COVID-19: Neutrophil extracellular trap(epmc).pdf DeepDyve Pubget Overpricing