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10.1016/j.bbi.2020.04.027

http://scihub22266oqcxt.onion/10.1016/j.bbi.2020.04.027
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32298803!7152874!32298803
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suck abstract from ncbi


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pmid32298803      Brain+Behav+Immun 2020 ; 87 (ä): 34-39
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  • Are we facing a crashing wave of neuropsychiatric sequelae of COVID-19? Neuropsychiatric symptoms and potential immunologic mechanisms #MMPMID32298803
  • Troyer EA; Kohn JN; Hong S
  • Brain Behav Immun 2020[Jul]; 87 (ä): 34-39 PMID32298803show ga
  • The coronavirus disease 19 (COVID-19) pandemic is a significant psychological stressor in addition to its tremendous impact on every facet of individuals' lives and organizations in virtually all social and economic sectors worldwide. Fear of illness and uncertainty about the future precipitate anxiety- and stress-related disorders, and several groups have rightfully called for the creation and dissemination of robust mental health screening and treatment programs for the general public and front-line healthcare workers. However, in addition to pandemic-associated psychological distress, the direct effects of the virus itself (several acute respiratory syndrome coronavirus; SARS-CoV-2), and the subsequent host immunologic response, on the human central nervous system (CNS) and related outcomes are unknown. We discuss currently available evidence of COVID-19 related neuropsychiatric sequelae while drawing parallels to past viral pandemic-related outcomes. Past pandemics have demonstrated that diverse types of neuropsychiatric symptoms, such as encephalopathy, mood changes, psychosis, neuromuscular dysfunction, or demyelinating processes, may accompany acute viral infection, or may follow infection by weeks, months, or longer in recovered patients. The potential mechanisms are also discussed, including viral and immunological underpinnings. Therefore, prospective neuropsychiatric monitoring of individuals exposed to SARS-CoV-2 at various points in the life course, as well as their neuroimmune status, are needed to fully understand the long-term impact of COVID-19, and to establish a framework for integrating psychoneuroimmunology into epidemiologic studies of pandemics.
  • |Acute Disease[MESH]
  • |Anxiety/etiology/immunology/psychology[MESH]
  • |Bacterial Translocation[MESH]
  • |Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Chronic Disease[MESH]
  • |Coronavirus Infections/complications/immunology/*psychology/therapy[MESH]
  • |Cytokine Release Syndrome/immunology/*psychology/therapy[MESH]
  • |Demyelinating Diseases/etiology/immunology/physiopathology/psychology[MESH]
  • |Depression/etiology/immunology/psychology[MESH]
  • |Humans[MESH]
  • |Immunologic Factors/adverse effects[MESH]
  • |Mental Disorders/etiology/immunology/*psychology[MESH]
  • |Mental Health[MESH]
  • |Nervous System Diseases/etiology/immunology/physiopathology/*psychology[MESH]
  • |Neurodegenerative Diseases/etiology/immunology/physiopathology/psychology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/complications/immunology/*psychology/therapy[MESH]
  • |Psychoneuroimmunology[MESH]
  • |Psychotic Disorders/etiology/immunology/psychology[MESH]
  • |Public Health[MESH]
  • |SARS-CoV-2[MESH]


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