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10.1016/j.jmii.2020.03.022 http://scihub22266oqcxt.onion/10.1016/j.jmii.2020.03.022 32265180!7138183!32265180     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Microbiol+Immunol+Infect 2021 ; 54 (2): 159-163 Nephropedia Template TP {{tp|p=32265180|t=2021. Genotype and phenotype of COVID-19: Their roles in pathogenesis |pdf=|usr=}}{{32265180}} gab.com Text Genotype and phenotype of COVID-19: Their roles in pathogenesis JO: J Microbiol Immunol Infect http://www.kidney.de/pget.php?&tw=1&pmid=32265180 #FOAvip COVID-19 is a novel coronavirus with an outbreak of unusual viral pneumonia in Wuhan, China, and then pandemic. Based on its phylogenetic relationships and genomic structures the COVID-19 belongs to genera Betacoronavirus. Human Betacoronaviruses (SARS-CoV-2, SARS-CoV, and MERS-CoV) have many similarities, but also have differences in their genomic and phenotypic structure that can influence their pathogenesis. COVID-19 is containing single-stranded (positive-sense) RNA associated with a nucleoprotein within a capsid comprised of matrix protein. A typical CoV contains at least six ORFs in its genome. All the structural and accessory proteins are translated from the sgRNAs of CoVs. Four main structural proteins are encoded by ORFs 10, 11 on the one-third of the genome near the 3'-terminus. The genetic and phenotypic structure of COVID-19 in pathogenesis is important. This article highlights the most important of these features compared to other Betacoronaviruses. Twit Text FOAVip Genotype and phenotype of COVID-19: Their roles in pathogenesis ????J Microbiol Immunol Infect http://www.kidney.de/pget.php?&tw=1&pmid=32265180 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32265180 #FOAvip English Wikipedia <ref>{{Cite pmid|32265180}}</ref> Genotype and phenotype of COVID-19: Their roles in pathogenesis #MMPMID32265180 Mousavizadeh L; Ghasemi S J Microbiol Immunol Infect 2021[Apr]; 54 (2): 159-163 PMID32265180show ga COVID-19 is a novel coronavirus with an outbreak of unusual viral pneumonia in Wuhan, China, and then pandemic. Based on its phylogenetic relationships and genomic structures the COVID-19 belongs to genera Betacoronavirus. Human Betacoronaviruses (SARS-CoV-2, SARS-CoV, and MERS-CoV) have many similarities, but also have differences in their genomic and phenotypic structure that can influence their pathogenesis. COVID-19 is containing single-stranded (positive-sense) RNA associated with a nucleoprotein within a capsid comprised of matrix protein. A typical CoV contains at least six ORFs in its genome. All the structural and accessory proteins are translated from the sgRNAs of CoVs. Four main structural proteins are encoded by ORFs 10, 11 on the one-third of the genome near the 3'-terminus. The genetic and phenotypic structure of COVID-19 in pathogenesis is important. This article highlights the most important of these features compared to other Betacoronaviruses. |Betacoronavirus/classification/genetics/pathogenicity[MESH] |COVID-19/etiology/*virology[MESH] |Genome, Viral[MESH] |Genotype[MESH] |Humans[MESH] |Pandemics[MESH] |Phenotype[MESH] |Phylogeny[MESH] |SARS-CoV-2/*genetics/pathogenicity/physiology[MESH] |Virulence/genetics[MESH]Genotype and phenotype of COVID-19: Their roles in pathogenesis (epmc).pdf DeepDyve Pubget Overpricing