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10.1038/s41581-020-0279-4 http://scihub22266oqcxt.onion/10.1038/s41581-020-0279-4 32246101!7118703!32246101     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Nat+Rev+Nephrol 2020 ; 16 (6): 305-307 Nephropedia Template TP {{tp|p=32246101|t=2020. Controversies of renin-angiotensin system inhibition during the COVID-19 pandemic |pdf=|usr=}}{{32246101}} gab.com Text Controversies of renin-angiotensin system inhibition during the COVID-19 pandemic JO: Nat Rev Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=32246101 #FOAvip The current COVID-19 pandemic is associated with unprecedented morbidity and mortality. Early reports suggested an association between disease severity and hypertension but did not account for sources of confounding. However, the responsible virus - SARS-CoV-2 - gains entry to host cells via angiotensin-converting enzyme 2 (ACE2), highlighting the need to understand the relationship between the virus and the renin-angiotensin system (RAS) and how this might be affected by RAS inhibitors. Twit Text FOAVip Controversies of renin-angiotensin system inhibition during the COVID-19 pandemic ????Nat Rev Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=32246101 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32246101 #FOAvip English Wikipedia <ref>{{Cite pmid|32246101}}</ref> Controversies of renin-angiotensin system inhibition during the COVID-19 pandemic #MMPMID32246101 South AM; Tomlinson L; Edmonston D; Hiremath S; Sparks MA Nat Rev Nephrol 2020[Jun]; 16 (6): 305-307 PMID32246101show ga The current COVID-19 pandemic is associated with unprecedented morbidity and mortality. Early reports suggested an association between disease severity and hypertension but did not account for sources of confounding. However, the responsible virus - SARS-CoV-2 - gains entry to host cells via angiotensin-converting enzyme 2 (ACE2), highlighting the need to understand the relationship between the virus and the renin-angiotensin system (RAS) and how this might be affected by RAS inhibitors. |Angiotensin-Converting Enzyme 2[MESH] |Betacoronavirus/*drug effects[MESH] |COVID-19[MESH] |COVID-19 Drug Treatment[MESH] |Coronavirus Infections/*drug therapy[MESH] |Enzyme Inhibitors/*therapeutic use[MESH] |Humans[MESH] |Pandemics[MESH] |Peptidyl-Dipeptidase A/metabolism[MESH] |Pneumonia, Viral/*drug therapy[MESH] |Renin-Angiotensin System/*drug effects[MESH] |SARS-CoV-2[MESH]Controversies of renin-angiotensin system inhibition during the COVID-(epmc).pdf DeepDyve Pubget Overpricing