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10.1038/s41580-020-0242-z

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32242128!7115351!32242128
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suck abstract from ncbi


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pmid32242128      Nat+Rev+Mol+Cell+Biol 2020 ; 21 (5): 247-248
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  • Mechano-genomic regulation of coronaviruses and its interplay with ageing #MMPMID32242128
  • Uhler C; Shivashankar GV
  • Nat Rev Mol Cell Biol 2020[May]; 21 (5): 247-248 PMID32242128show ga
  • Coronaviruses employ various nuclear signalling mechanisms for their propagation. Here we link replication of coronaviruses, including SARS-CoV-2, the coronavirus disease 2019 (COVID-19)-causing virus, with cytoskeleton-dependent signalling and 3D genome organization. This link could provide novel insights into the high coronavirus pathogenicity in certain cellular states, particularly those associated with ageing.
  • |Aging/*physiology[MESH]
  • |Betacoronavirus/pathogenicity/*physiology[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/pathology[MESH]
  • |Cytoskeleton/metabolism/*virology[MESH]
  • |Genome, Viral[MESH]
  • |Host-Pathogen Interactions/*physiology[MESH]
  • |Humans[MESH]
  • |NF-kappa B/metabolism[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/pathology[MESH]
  • |SARS-CoV-2[MESH]
  • |Signal Transduction[MESH]


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