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10.1016/j.ekir.2019.04.013 http://scihub22266oqcxt.onion/10.1016/j.ekir.2019.04.013 31317113!6611949!31317113     free     free     free       Kidney+Int+Rep 2019 ; 4 (7): 917-922 Nephropedia Template TP {{tp|p=31317113|t=2019. Fibrillary Glomerulonephritis: An Update |pdf=|usr=}}{{31317113}} gab.com Text Fibrillary Glomerulonephritis: An Update JO: Kidney Int Rep http://www.kidney.de/pget.php?&tw=1&pmid=31317113 #FOAvip Fibrillary glomerulonephritis (FGN) is a rare proliferative form of glomerular disease characterized by randomly oriented fibrillar deposits with a mean diameter of 20 nm. By immunofluorescence (IF), the deposits stain for IgG, C3, and kappa and lambda light chains, suggesting that the fibrils may be composed of antigen-antibody immune complexes. A recent major advance in our understanding of the pathogenesis of FGN resulted from the discovery that a major component of the fibrils is DNA-J heat-shock protein family member B9 (DNAJB9), and immunohistochemical staining for DNAJB9 now makes it possible to diagnose FGN in the absence of ultrastructural evaluation. FGN has a poor prognosis, treatment options are currently limited, and transplant recurrence is not uncommon. Twit Text FOAVip Fibrillary Glomerulonephritis: An Update ????Kidney Int Rep http://www.kidney.de/pget.php?&tw=1&pmid=31317113 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=31317113 #FOAvip English Wikipedia <ref>{{Cite pmid|31317113}}</ref> Fibrillary Glomerulonephritis: An Update #MMPMID31317113 Rosenstock JL; Markowitz GS Kidney Int Rep 2019[Jul]; 4 (7): 917-922 PMID31317113show ga Fibrillary glomerulonephritis (FGN) is a rare proliferative form of glomerular disease characterized by randomly oriented fibrillar deposits with a mean diameter of 20 nm. By immunofluorescence (IF), the deposits stain for IgG, C3, and kappa and lambda light chains, suggesting that the fibrils may be composed of antigen-antibody immune complexes. A recent major advance in our understanding of the pathogenesis of FGN resulted from the discovery that a major component of the fibrils is DNA-J heat-shock protein family member B9 (DNAJB9), and immunohistochemical staining for DNAJB9 now makes it possible to diagnose FGN in the absence of ultrastructural evaluation. FGN has a poor prognosis, treatment options are currently limited, and transplant recurrence is not uncommon. ?Fibrillary Glomerulonephritis: An Update (epmc).pdf DeepDyve Pubget Overpricing