Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1289/ehp.7826181 http://scihub22266oqcxt.onion/10.1289/ehp.7826181 31282!1637252!31282     free     free     free       Environ+Health+Perspect 1978 ; 26 (ä): 181-91 Nephropedia Template TP {{tp|p=31282|t=1978. Cardiac lesions induced by chemicals |pdf=|usr=}}{{31282}} gab.com Text Cardiac lesions induced by chemicals JO: Environ Health Perspect http://www.kidney.de/pget.php?&tw=1&pmid=31282 #FOAvip Chemically induced cardiomyopathies are frequently the consequences of a cardiac metabolic imbalance brought about by exaggerated functional affects. The infarctlike lesions induced by adrenergic beta-receptor stimulants and the vasodilating antihypertensives serve as examples of this phenomenon. Direct cardiotoxic mechanisms not related to cardiovascular functional effects are responsible for another class of toxic cardiomyopathy. An example of this is the cardiomyopathy produced by the anthracycline antineoplastic agents. The pathogenesis, morphological changes and toxicologic features of these cardiomyopathies are described with particular reference to their detection in preclinical toxicity studies. Twit Text FOAVip Cardiac lesions induced by chemicals ????Environ Health Perspect http://www.kidney.de/pget.php?&tw=1&pmid=31282 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=31282 #FOAvip English Wikipedia <ref>{{Cite pmid|31282}}</ref> Cardiac lesions induced by chemicals #MMPMID31282 Balazs T; Ferrans VJ Environ Health Perspect 1978[Oct]; 26 (ä): 181-91 PMID31282show ga Chemically induced cardiomyopathies are frequently the consequences of a cardiac metabolic imbalance brought about by exaggerated functional affects. The infarctlike lesions induced by adrenergic beta-receptor stimulants and the vasodilating antihypertensives serve as examples of this phenomenon. Direct cardiotoxic mechanisms not related to cardiovascular functional effects are responsible for another class of toxic cardiomyopathy. An example of this is the cardiomyopathy produced by the anthracycline antineoplastic agents. The pathogenesis, morphological changes and toxicologic features of these cardiomyopathies are described with particular reference to their detection in preclinical toxicity studies. |Acute Disease[MESH] |Adrenergic beta-Agonists/adverse effects[MESH] |Animals[MESH] |Antihypertensive Agents/adverse effects[MESH] |Cardiomyopathies/chemically induced/pathology[MESH] |Cardiovascular System/drug effects[MESH] |Chronic Disease[MESH] |Daunorubicin/adverse effects[MESH] |Doxorubicin/adverse effects[MESH] |Drug Evaluation, Preclinical[MESH] |Heart Diseases/*chemically induced/pathology[MESH] |Humans[MESH]Cardiac lesions induced by chemicals (epmc).pdf DeepDyve Pubget Overpricing