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10.1152/ajprenal.00604.2018 http://scihub22266oqcxt.onion/10.1152/ajprenal.00604.2018 31215804!6732451!31215804     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Physiol+Renal+Physiol 2019 ; 317 (2): F444-F455 Nephropedia Template TP {{tp|p=31215804|t=2019. Effects of reactive oxygen species on renal tubular transport |pdf=|usr=}}{{31215804}} gab.com Text Effects of reactive oxygen species on renal tubular transport JO: Am J Physiol Renal Physiol http://www.kidney.de/pget.php?&tw=1&pmid=31215804 #FOAvip Reactive oxygen species (ROS) play a critical role in regulating nephron transport both via transcellular and paracellular pathways under physiological and pathological circumstances. Here, we review the progress made in the past ~10 yr in understanding how ROS regulate solute and water transport in individual nephron segments. Our knowledge in this field is still rudimentary, with basic information lacking. This is most obvious when looking at the reported disparate effects of superoxide ([Formula: see text]) and H(2)O(2) on proximal nephron transport, where there are no easy explanations as to how to reconcile the data. Similarly, we know almost nothing about the regulation of transport in thin descending and ascending limbs, information that is likely critical to understanding the urine concentrating mechanism. In the thick ascending limb, there is general agreement that ROS enhance transcellular reabsorption of NaCl, but we know very little about their effects on the paracellular pathway and therefore Ca(2+) and Mg(2+) transport. In the distal convoluted tubule, precious little is known. In the collecting duct, there is general agreement that ROS stimulate the epithelial Na(+) channel. Twit Text FOAVip Effects of reactive oxygen species on renal tubular transport ????Am J Physiol Renal Physiol http://www.kidney.de/pget.php?&tw=1&pmid=31215804 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=31215804 #FOAvip English Wikipedia <ref>{{Cite pmid|31215804}}</ref> Effects of reactive oxygen species on renal tubular transport #MMPMID31215804 Gonzalez-Vicente A; Hong N; Garvin JL Am J Physiol Renal Physiol 2019[Aug]; 317 (2): F444-F455 PMID31215804show ga Reactive oxygen species (ROS) play a critical role in regulating nephron transport both via transcellular and paracellular pathways under physiological and pathological circumstances. Here, we review the progress made in the past ~10 yr in understanding how ROS regulate solute and water transport in individual nephron segments. Our knowledge in this field is still rudimentary, with basic information lacking. This is most obvious when looking at the reported disparate effects of superoxide ([Formula: see text]) and H(2)O(2) on proximal nephron transport, where there are no easy explanations as to how to reconcile the data. Similarly, we know almost nothing about the regulation of transport in thin descending and ascending limbs, information that is likely critical to understanding the urine concentrating mechanism. In the thick ascending limb, there is general agreement that ROS enhance transcellular reabsorption of NaCl, but we know very little about their effects on the paracellular pathway and therefore Ca(2+) and Mg(2+) transport. In the distal convoluted tubule, precious little is known. In the collecting duct, there is general agreement that ROS stimulate the epithelial Na(+) channel. |Animals[MESH] |Biological Transport[MESH] |Humans[MESH] |Kidney Tubules/*metabolism[MESH] |Reactive Oxygen Species/*metabolism[MESH]Effects of reactive oxygen species on renal tubular transport (epmc).pdf DeepDyve Pubget Overpricing