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10.1016/j.micinf.2018.03.005 http://scihub22266oqcxt.onion/10.1016/j.micinf.2018.03.005 29679740!7110448!29679740     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Microbes+Infect 2018 ; 20 (9-10): 560-569 Nephropedia Template TP {{tp|p=29679740|t=2018. Dissecting host cell death programs in the pathogenesis of influenza |pdf=|usr=}}{{29679740}} gab.com Text Dissecting host cell death programs in the pathogenesis of influenza JO: Microbes Infect http://www.kidney.de/pget.php?&tw=1&pmid=29679740 #FOAvip Influenza A virus (IAV) is a pulmonary pathogen, responsible for significant yearly morbidity and mortality. Due to the absence of highly effective antiviral therapies and vaccine, as well as the constant threat of an emerging pandemic strain, there is considerable need to better understand the host-pathogen interactions and the factors that dictate a protective versus detrimental immune response to IAV. Even though evidence of IAV-induced cell death in human pulmonary epithelial and immune cells has been observed for almost a century, very little is known about the consequences of cell death on viral pathogenesis. Recent study indicates that both the type of cell death program and its kinetics have major implications on host defense and survival. In this review, we discuss advances in our understanding of cell death programs during influenza virus infection, in hopes of fostering new areas of investigation for targeted clinical intervention. Twit Text FOAVip Dissecting host cell death programs in the pathogenesis of influenza ????Microbes Infect http://www.kidney.de/pget.php?&tw=1&pmid=29679740 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29679740 #FOAvip English Wikipedia <ref>{{Cite pmid|29679740}}</ref> Dissecting host cell death programs in the pathogenesis of influenza #MMPMID29679740 Downey J; Pernet E; Coulombe F; Divangahi M Microbes Infect 2018[Oct]; 20 (9-10): 560-569 PMID29679740show ga Influenza A virus (IAV) is a pulmonary pathogen, responsible for significant yearly morbidity and mortality. Due to the absence of highly effective antiviral therapies and vaccine, as well as the constant threat of an emerging pandemic strain, there is considerable need to better understand the host-pathogen interactions and the factors that dictate a protective versus detrimental immune response to IAV. Even though evidence of IAV-induced cell death in human pulmonary epithelial and immune cells has been observed for almost a century, very little is known about the consequences of cell death on viral pathogenesis. Recent study indicates that both the type of cell death program and its kinetics have major implications on host defense and survival. In this review, we discuss advances in our understanding of cell death programs during influenza virus infection, in hopes of fostering new areas of investigation for targeted clinical intervention. |*Cell Death[MESH] |Alveolar Epithelial Cells/pathology[MESH] |Animals[MESH] |Host-Pathogen Interactions/*immunology[MESH] |Humans[MESH] |Influenza A virus/*physiology[MESH] |Influenza, Human/*immunology[MESH] |Kinetics[MESH] |Macrophages, Alveolar/pathology[MESH]Dissecting host cell death programs in the pathogenesis of influenza (epmc).pdf DeepDyve Pubget Overpricing