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10.1186/s12929-018-0436-2 http://scihub22266oqcxt.onion/10.1186/s12929-018-0436-2 29650017!5898010!29650017     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29650017&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Biomed+Sci 2018 ; 25 (1): 35 Nephropedia Template TP {{tp|p=29650017|t=2018. Toll-like receptors in lupus nephritis |pdf=|usr=}}{{29650017}} gab.com Text Toll-like receptors in lupus nephritis JO: J Biomed Sci http://www.kidney.de/pget.php?&tw=1&pmid=29650017 #FOAvip The pathogenesis of systemic autoimmune diseases such as systemic lupus erythematosus (SLE) is based on the loss of self-tolerance against ubiquitous autoantigens involving all mechanisms of adaptive immunity. However, data accumulating over the last decade imply an important role also for numerous elements of innate immunity, namely the Toll-like receptors in the pathogenesis of SLE. Here we discuss their role in the most common organ complication of SLE, i.e. lupus nephritis. We summarize experimental and clinical data on the expression and functional contribution of the Toll-like receptors in immune complex glomerulonephritis, and intrarenal inflammation. Based on these discoveries Toll-like receptors are evolving as therapeutic targets for the treatment of SLE and lupus nephritis. Twit Text FOAVip Toll-like receptors in lupus nephritis ????J Biomed Sci http://www.kidney.de/pget.php?&tw=1&pmid=29650017 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29650017 #FOAvip English Wikipedia <ref>{{Cite pmid|29650017}}</ref> Toll-like receptors in lupus nephritis #MMPMID29650017 Devarapu SK; Anders HJ J Biomed Sci 2018[Apr]; 25 (1): 35 PMID29650017show ga The pathogenesis of systemic autoimmune diseases such as systemic lupus erythematosus (SLE) is based on the loss of self-tolerance against ubiquitous autoantigens involving all mechanisms of adaptive immunity. However, data accumulating over the last decade imply an important role also for numerous elements of innate immunity, namely the Toll-like receptors in the pathogenesis of SLE. Here we discuss their role in the most common organ complication of SLE, i.e. lupus nephritis. We summarize experimental and clinical data on the expression and functional contribution of the Toll-like receptors in immune complex glomerulonephritis, and intrarenal inflammation. Based on these discoveries Toll-like receptors are evolving as therapeutic targets for the treatment of SLE and lupus nephritis. |Humans[MESH] |Inflammation/genetics/immunology/therapy[MESH] |Lupus Nephritis/genetics/immunology/*therapy[MESH]Toll-like receptors in lupus nephritis (epmc).pdf DeepDyve Pubget Overpricing