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10.3390/nu9020112 http://scihub22266oqcxt.onion/10.3390/nu9020112 28178182!5331543!28178182     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Nutrients 2017 ; 9 (2): ä Nephropedia Template TP {{tp|p=28178182|t=2017. Effects of Magnesium on the Phosphate Toxicity in Chronic Kidney Disease: Time for Intervention Studies |pdf=|usr=}}{{28178182}} gab.com Text Effects of Magnesium on the Phosphate Toxicity in Chronic Kidney Disease: Time for Intervention Studies JO: Nutrients http://www.kidney.de/pget.php?&tw=1&pmid=28178182 #FOAvip Magnesium, an essential mineral for human health, plays a pivotal role in the cardiovascular system. Epidemiological studies in the general population have found an association between lower dietary magnesium intake and an elevated risk of cardiovascular events. In addition, magnesium supplementation was shown to improve blood pressure control, insulin sensitivity, and endothelial function. The relationship between magnesium and cardiovascular prognosis among patients with chronic kidney disease (CKD) has been increasingly investigated as it is becoming evident that magnesium can inhibit vascular calcification, a prominent risk of cardiovascular events, which commonly occurs in CKD patients. Cohort studies in patients receiving dialysis have shown a lower serum magnesium level as a significant risk for cardiovascular mortality. Interestingly, the cardiovascular mortality risk associated with hyperphosphatemia is alleviated among those with high serum magnesium levels, consistent with in vitro evidence that magnesium inhibits high-phosphate induced calcification of vascular smooth muscle cells. Furthermore, a harmful effect of high phosphate on the progression of CKD is also attenuated among those with high serum magnesium levels. The potential usefulness of magnesium as a remedy for phosphate toxicity should be further explored by future intervention studies. Twit Text FOAVip Effects of Magnesium on the Phosphate Toxicity in Chronic Kidney Disease: Time for Intervention Studies ????Nutrients http://www.kidney.de/pget.php?&tw=1&pmid=28178182 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28178182 #FOAvip English Wikipedia <ref>{{Cite pmid|28178182}}</ref> Effects of Magnesium on the Phosphate Toxicity in Chronic Kidney Disease: Time for Intervention Studies #MMPMID28178182 Sakaguchi Y; Hamano T; Isaka Y Nutrients 2017[Feb]; 9 (2): ä PMID28178182show ga Magnesium, an essential mineral for human health, plays a pivotal role in the cardiovascular system. Epidemiological studies in the general population have found an association between lower dietary magnesium intake and an elevated risk of cardiovascular events. In addition, magnesium supplementation was shown to improve blood pressure control, insulin sensitivity, and endothelial function. The relationship between magnesium and cardiovascular prognosis among patients with chronic kidney disease (CKD) has been increasingly investigated as it is becoming evident that magnesium can inhibit vascular calcification, a prominent risk of cardiovascular events, which commonly occurs in CKD patients. Cohort studies in patients receiving dialysis have shown a lower serum magnesium level as a significant risk for cardiovascular mortality. Interestingly, the cardiovascular mortality risk associated with hyperphosphatemia is alleviated among those with high serum magnesium levels, consistent with in vitro evidence that magnesium inhibits high-phosphate induced calcification of vascular smooth muscle cells. Furthermore, a harmful effect of high phosphate on the progression of CKD is also attenuated among those with high serum magnesium levels. The potential usefulness of magnesium as a remedy for phosphate toxicity should be further explored by future intervention studies. |Animals[MESH] |Blood Pressure/drug effects[MESH] |Cardiovascular Diseases/blood/prevention & control[MESH] |Cohort Studies[MESH] |Disease Models, Animal[MESH] |Disease Progression[MESH] |Humans[MESH] |Hyperphosphatemia/blood/drug therapy[MESH] |Insulin Resistance[MESH] |Magnesium/*administration & dosage[MESH] |Phosphates/*toxicity[MESH] |Renal Dialysis[MESH] |Renal Insufficiency, Chronic/*drug therapy/etiology[MESH] |Risk Factors[MESH]Effects of Magnesium on the Phosphate Toxicity in Chronic Kidney Disea(epmc).pdf DeepDyve Pubget Overpricing