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10.1002/jmv.1890270302 http://scihub22266oqcxt.onion/10.1002/jmv.1890270302 2786050!ä!2786050 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=2786050&cmd=llinks): Failed to open stream: HTTP request failed! 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Regulation of lymphocyte proliferation after influenza virus infection of human mononuclear leukocytes |pdf=|usr=}}{{2786050}} gab.com Text Regulation of lymphocyte proliferation after influenza virus infection of human mononuclear leukocytes JO: J Med Virol http://www.kidney.de/pget.php?&tw=1&pmid=2786050 #FOAvip Previous studies demonstrated that in vitro infection with influenza A viruses altered several functions of human monocytes-macrophages but did not detectably alter functions of human lymphocytes. However, both types of cells were infected, as determined by production and surface expression of viral antigens. In the current studies, human mononuclear leukocytes were infected in vitro and assayed for both influenza virus-induced proliferation and mitogen (phytohemagglutinin [PHAl)-induced proliferation, as well as for ability to stimulate proliferative responses by normal autologous leukocytes. The leukocytes showed proliferation in response to the infectious virus, but concomitant depressed proliferative responses to PHA. Coculture experiments suggested suppression of PHA-induced responses by the virus-infected cells. However, upon coculture with fresh autologous leukocytes (without PHA stimulation), both virus-infected macrophages and virus-infected lymphocytes induced autologous lymphocyte proliferative responses. Altered proliferative responses to mitogen stimulation after exposure to the virus were not due to diminished interleukin-1 production or diminished expression of HLA-DR by monocytes-macrophages. The expression of influenza virus antigens and resulting induction of autologous proliferative responses, combined with depressed mitogen-induced proliferation, may be important in human antiviral defense. Twit Text FOAVip Regulation of lymphocyte proliferation after influenza virus infection of human mononuclear leukocytes ????J Med Virol http://www.kidney.de/pget.php?&tw=1&pmid=2786050 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=2786050 #FOAvip English Wikipedia <ref>{{Cite pmid|2786050}}</ref> Regulation of lymphocyte proliferation after influenza virus infection of human mononuclear leukocytes #MMPMID2786050 Roberts NJ Jr; Nichols JE J Med Virol 1989[Mar]; 27 (3): 179-87 PMID2786050show ga Previous studies demonstrated that in vitro infection with influenza A viruses altered several functions of human monocytes-macrophages but did not detectably alter functions of human lymphocytes. However, both types of cells were infected, as determined by production and surface expression of viral antigens. In the current studies, human mononuclear leukocytes were infected in vitro and assayed for both influenza virus-induced proliferation and mitogen (phytohemagglutinin [PHAl)-induced proliferation, as well as for ability to stimulate proliferative responses by normal autologous leukocytes. The leukocytes showed proliferation in response to the infectious virus, but concomitant depressed proliferative responses to PHA. Coculture experiments suggested suppression of PHA-induced responses by the virus-infected cells. However, upon coculture with fresh autologous leukocytes (without PHA stimulation), both virus-infected macrophages and virus-infected lymphocytes induced autologous lymphocyte proliferative responses. Altered proliferative responses to mitogen stimulation after exposure to the virus were not due to diminished interleukin-1 production or diminished expression of HLA-DR by monocytes-macrophages. The expression of influenza virus antigens and resulting induction of autologous proliferative responses, combined with depressed mitogen-induced proliferation, may be important in human antiviral defense. |*Lymphocyte Activation/drug effects[MESH] |Adult[MESH] |Antigen-Presenting Cells/immunology[MESH] |Antigens, Viral/immunology[MESH] |Cells, Cultured[MESH] |Concanavalin A/pharmacology[MESH] |Female[MESH] |HLA-DR Antigens/biosynthesis[MESH] |Humans[MESH] |Influenza A virus/*physiology[MESH] |Interleukin-1/biosynthesis[MESH] |Leukocytes, Mononuclear/*cytology/immunology/microbiology[MESH] |Lymphocytes/cytology/immunology/microbiology[MESH] |Macrophages/immunology/microbiology[MESH] |Male[MESH] |Monocytes/microbiology[MESH]Regulation of lymphocyte proliferation after influenza virus infection(epmc).pdf DeepDyve Pubget Overpricing