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10.1111/micc.12329 http://scihub22266oqcxt.onion/10.1111/micc.12329 27809396!5404942!27809396     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27809396&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Microcirculation 2017 ; 24 (3): ? Nephropedia Template TP {{tp|p=27809396|t=2017. Redox regulation of transient receptor potential channels in the endothelium |pdf=|usr=}}{{27809396}} gab.com Text Redox regulation of transient receptor potential channels in the endothelium JO: Microcirculation http://www.kidney.de/pget.php?&tw=1&pmid=27809396 #FOAvip ROS and RNS are important mediators of signaling pathways in the endothelium. Specific members of the TRP superfamily of cation channels act as important Ca(2+) influx pathways in endothelial cells and are involved in endothelium-dependent vasodilation, regulation of barrier permeability, and angiogenesis. ROS and RNS can modulate the activity of certain TRP channels mainly by modifying specific cysteine residues or by stimulating the production of second messengers. In this review, we highlight the recent literature describing redox regulation of TRP channel activity in endothelial cells as well as the physiological importance of these pathways and implication for cardiovascular diseases. Twit Text FOAVip Redox regulation of transient receptor potential channels in the endothelium ????Microcirculation http://www.kidney.de/pget.php?&tw=1&pmid=27809396 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27809396 #FOAvip English Wikipedia <ref>{{Cite pmid|27809396}}</ref> Redox regulation of transient receptor potential channels in the endothelium #MMPMID27809396 Pires PW; Earley S Microcirculation 2017[Apr]; 24 (3): ? PMID27809396show ga ROS and RNS are important mediators of signaling pathways in the endothelium. Specific members of the TRP superfamily of cation channels act as important Ca(2+) influx pathways in endothelial cells and are involved in endothelium-dependent vasodilation, regulation of barrier permeability, and angiogenesis. ROS and RNS can modulate the activity of certain TRP channels mainly by modifying specific cysteine residues or by stimulating the production of second messengers. In this review, we highlight the recent literature describing redox regulation of TRP channel activity in endothelial cells as well as the physiological importance of these pathways and implication for cardiovascular diseases. |*Oxidation-Reduction[MESH] |Animals[MESH] |Endothelium, Vascular/*metabolism[MESH] |Humans[MESH] |Reactive Nitrogen Species/metabolism[MESH] |Reactive Oxygen Species/metabolism[MESH] |Second Messenger Systems[MESH] |Signal Transduction[MESH]Redox regulation of transient receptor potential channels in the endot(epmc).pdf DeepDyve Pubget Overpricing