Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.kint.2016.07.021 http://scihub22266oqcxt.onion/10.1016/j.kint.2016.07.021 27650730!ä!27650730 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Kidney+Int 2016 ; 90 (6): 1321-1331 Nephropedia Template TP {{tp|p=27650730|t=2016. Collapsing glomerulopathy is common in the setting of thrombotic microangiopathy of the native kidney |pdf=|usr=}}{{27650730}} gab.com Text Collapsing glomerulopathy is common in the setting of thrombotic microangiopathy of the native kidney JO: Kidney Int http://www.kidney.de/pget.php?&tw=1&pmid=27650730 #FOAvip Thrombotic microangiopathy (TMA) is a poorly recognized cause of collapsing glomerulopathy. The frequency and significance of collapsing glomerulopathy associated with renal TMA have not been specifically studied in native kidney biopsy specimens. Here we retrospectively documented clinicopathologic features of 53 patients with histologically proven TMA in the native kidney, with special emphasis on changes due to focal segmental glomerulosclerosis (FSGS). Histological TMA was related to hypertensive nephropathy in 21 patients, genetic complement abnormalities in 9, drugs in 7, and to other causes in 16 patients. Almost half (26 patients) presented with arteriolar, 6 with glomerular, and 21 with mixed TMA. Using the Columbia classification system for the 53 patients with histological TMA, 33 had concurrent FSGS lesions with collapsing glomerulopathy the dominant variant in 19 patients (58% of the FSGS cases), not otherwise specified in 9 patients, cellular in 3, and perihilar or tip lesions in 1 patient each. The presence of FSGS was associated with a poor renal prognosis, with no prognostic difference between collapsing glomerulopathy and other FSGS variants. Thus, collapsing glomerulopathy is frequently found in native kidney biopsies with TMA, suggesting that endothelial injury may play an important role in the pathophysiology of FSGS. Twit Text FOAVip Collapsing glomerulopathy is common in the setting of thrombotic microangiopathy of the native kidney ????Kidney Int http://www.kidney.de/pget.php?&tw=1&pmid=27650730 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27650730 #FOAvip English Wikipedia <ref>{{Cite pmid|27650730}}</ref> Collapsing glomerulopathy is common in the setting of thrombotic microangiopathy of the native kidney #MMPMID27650730 Buob D; Decambron M; Gnemmi V; Frimat M; Hoffmann M; Azar R; Gheerbrant JD; Guincestre T; Noel C; Copin MC; Glowacki F Kidney Int 2016[Dec]; 90 (6): 1321-1331 PMID27650730show ga Thrombotic microangiopathy (TMA) is a poorly recognized cause of collapsing glomerulopathy. The frequency and significance of collapsing glomerulopathy associated with renal TMA have not been specifically studied in native kidney biopsy specimens. Here we retrospectively documented clinicopathologic features of 53 patients with histologically proven TMA in the native kidney, with special emphasis on changes due to focal segmental glomerulosclerosis (FSGS). Histological TMA was related to hypertensive nephropathy in 21 patients, genetic complement abnormalities in 9, drugs in 7, and to other causes in 16 patients. Almost half (26 patients) presented with arteriolar, 6 with glomerular, and 21 with mixed TMA. Using the Columbia classification system for the 53 patients with histological TMA, 33 had concurrent FSGS lesions with collapsing glomerulopathy the dominant variant in 19 patients (58% of the FSGS cases), not otherwise specified in 9 patients, cellular in 3, and perihilar or tip lesions in 1 patient each. The presence of FSGS was associated with a poor renal prognosis, with no prognostic difference between collapsing glomerulopathy and other FSGS variants. Thus, collapsing glomerulopathy is frequently found in native kidney biopsies with TMA, suggesting that endothelial injury may play an important role in the pathophysiology of FSGS. |Adult[MESH] |Aged[MESH] |Female[MESH] |France/epidemiology[MESH] |Glomerulosclerosis, Focal Segmental/epidemiology/*etiology/pathology[MESH] |Humans[MESH] |Kidney/*pathology[MESH] |Male[MESH] |Middle Aged[MESH] |Prevalence[MESH] |Retrospective Studies[MESH] |Thrombotic Microangiopathies/*complications[MESH]Collapsing glomerulopathy is common in the setting of thrombotic micro(epmc).pdf DeepDyve Pubget Overpricing