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suck abstract from ncbi


10.1101/cshperspect.a023473

http://scihub22266oqcxt.onion/10.1101/cshperspect.a023473
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26292986!4588136!26292986
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suck abstract from ncbi

pmid26292986      Cold+Spring+Harb+Perspect+Med 2015 ; 5 (10): ?
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  • Molecular Mechanisms of Preeclampsia #MMPMID26292986
  • Hod T; Cerdeira AS; Karumanchi SA
  • Cold Spring Harb Perspect Med 2015[Aug]; 5 (10): ? PMID26292986show ga
  • Preeclampsia is a pregnancy-specific disease characterized by new onset hypertension and proteinuria after 20 wk of gestation. It is a leading cause of maternal and fetal morbidity and mortality worldwide. Exciting discoveries in the last decade have contributed to a better understanding of the molecular basis of this disease. Epidemiological, experimental, and therapeutic studies from several laboratories have provided compelling evidence that an antiangiogenic state owing to alterations in circulating angiogenic factors leads to preeclampsia. In this review, we highlight the role of key circulating antiangiogenic factors as pathogenic biomarkers and in the development of novel therapies for preeclampsia.
  • |Angiogenesis Inducing Agents/metabolism/therapeutic use[MESH]
  • |Biomarkers/*metabolism[MESH]
  • |Female[MESH]
  • |Humans[MESH]
  • |Placenta Diseases/diagnosis/drug therapy/etiology[MESH]
  • |Placenta/metabolism[MESH]
  • |Pre-Eclampsia/diagnosis/drug therapy/*etiology[MESH]
  • |Pregnancy[MESH]
  • |Prenatal Diagnosis/methods[MESH]
  • |Signal Transduction/physiology[MESH]
  • |Vascular Endothelial Growth Factor A/physiology[MESH]


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