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10.1101/cshperspect.a023473 http://scihub22266oqcxt.onion/10.1101/cshperspect.a023473 26292986!4588136!26292986     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26292986&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Cold+Spring+Harb+Perspect+Med 2015 ; 5 (10): ? Nephropedia Template TP {{tp|p=26292986|t=2015. Molecular Mechanisms of Preeclampsia |pdf=|usr=}}{{26292986}} gab.com Text Molecular Mechanisms of Preeclampsia JO: Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=26292986 #FOAvip Preeclampsia is a pregnancy-specific disease characterized by new onset hypertension and proteinuria after 20 wk of gestation. It is a leading cause of maternal and fetal morbidity and mortality worldwide. Exciting discoveries in the last decade have contributed to a better understanding of the molecular basis of this disease. Epidemiological, experimental, and therapeutic studies from several laboratories have provided compelling evidence that an antiangiogenic state owing to alterations in circulating angiogenic factors leads to preeclampsia. In this review, we highlight the role of key circulating antiangiogenic factors as pathogenic biomarkers and in the development of novel therapies for preeclampsia. Twit Text FOAVip Molecular Mechanisms of Preeclampsia ????Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=26292986 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26292986 #FOAvip English Wikipedia <ref>{{Cite pmid|26292986}}</ref> Molecular Mechanisms of Preeclampsia #MMPMID26292986 Hod T; Cerdeira AS; Karumanchi SA Cold Spring Harb Perspect Med 2015[Aug]; 5 (10): ? PMID26292986show ga Preeclampsia is a pregnancy-specific disease characterized by new onset hypertension and proteinuria after 20 wk of gestation. It is a leading cause of maternal and fetal morbidity and mortality worldwide. Exciting discoveries in the last decade have contributed to a better understanding of the molecular basis of this disease. Epidemiological, experimental, and therapeutic studies from several laboratories have provided compelling evidence that an antiangiogenic state owing to alterations in circulating angiogenic factors leads to preeclampsia. In this review, we highlight the role of key circulating antiangiogenic factors as pathogenic biomarkers and in the development of novel therapies for preeclampsia. |Angiogenesis Inducing Agents/metabolism/therapeutic use[MESH] |Biomarkers/*metabolism[MESH] |Female[MESH] |Humans[MESH] |Placenta Diseases/diagnosis/drug therapy/etiology[MESH] |Placenta/metabolism[MESH] |Pre-Eclampsia/diagnosis/drug therapy/*etiology[MESH] |Pregnancy[MESH] |Prenatal Diagnosis/methods[MESH] |Signal Transduction/physiology[MESH] |Vascular Endothelial Growth Factor A/physiology[MESH]Molecular Mechanisms of Preeclampsia (epmc).pdf DeepDyve Pubget Overpricing