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10.1111/imm.12507 http://scihub22266oqcxt.onion/10.1111/imm.12507 26190495!4582961!26190495     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Immunology 2015 ; 146 (2): 194-205 Nephropedia Template TP {{tp|p=26190495|t=2015. Inflammation-associated genes: risks and benefits to Foxp3+ regulatory T-cell function |pdf=|usr=}}{{26190495}} gab.com Text Inflammation-associated genes: risks and benefits to Foxp3+ regulatory T-cell function JO: Immunology http://www.kidney.de/pget.php?&tw=1&pmid=26190495 #FOAvip Foxp3(+) regulatory T (Treg) cells prevent the development of autoimmunity and immunopathology, as well as maintaining homeostasis and tolerance to commensal microorganisms. The suppressive activity of Treg cells is their defining characteristic, generating great interest in their therapeutic potential. However, suppressive and effector functions are not entirely exclusive. Considerable evidence points to the ability of supposedly anti-inflammatory Foxp3-expressing Treg cells to also express transcription factors that have been characterized as cardinal drivers of T effector cell function. We will consider the mounting evidence that Treg cells can function in non-suppressive capacities and review the impetus for this functional change, its relevance to developing immune and autoimmune responses and its significance to the development of Treg-based therapies. Twit Text FOAVip Inflammation-associated genes: risks and benefits to Foxp3+ regulatory T-cell function ????Immunology http://www.kidney.de/pget.php?&tw=1&pmid=26190495 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26190495 #FOAvip English Wikipedia <ref>{{Cite pmid|26190495}}</ref> Inflammation-associated genes: risks and benefits to Foxp3+ regulatory T-cell function #MMPMID26190495 O'Connor RA; Anderton SM Immunology 2015[Oct]; 146 (2): 194-205 PMID26190495show ga Foxp3(+) regulatory T (Treg) cells prevent the development of autoimmunity and immunopathology, as well as maintaining homeostasis and tolerance to commensal microorganisms. The suppressive activity of Treg cells is their defining characteristic, generating great interest in their therapeutic potential. However, suppressive and effector functions are not entirely exclusive. Considerable evidence points to the ability of supposedly anti-inflammatory Foxp3-expressing Treg cells to also express transcription factors that have been characterized as cardinal drivers of T effector cell function. We will consider the mounting evidence that Treg cells can function in non-suppressive capacities and review the impetus for this functional change, its relevance to developing immune and autoimmune responses and its significance to the development of Treg-based therapies. |Animals[MESH] |Cytokines/genetics/*immunology/metabolism[MESH] |Forkhead Transcription Factors/*immunology/metabolism[MESH] |Genetic Markers[MESH] |Genetic Predisposition to Disease[MESH] |Humans[MESH] |Immunophenotyping[MESH] |Inflammation Mediators/*immunology/metabolism[MESH] |Inflammation/genetics/*immunology/metabolism/therapy[MESH] |Phenotype[MESH] |Receptors, Cytokine/genetics/*immunology/metabolism[MESH] |T-Lymphocytes, Helper-Inducer/immunology/metabolism[MESH] |T-Lymphocytes, Regulatory/*immunology/metabolism[MESH]Inflammation-associated genes: risks and benefits to Foxp3+ regulatory(epmc).pdf DeepDyve Pubget Overpricing