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10.1007/s13181-015-0483-y http://scihub22266oqcxt.onion/10.1007/s13181-015-0483-y 26159649!4781799!26159649     free     free     free       J+Med+Toxicol 2016 ; 12 (1): 121-9 Nephropedia Template TP {{tp|p=26159649|t=2016. A Literature Review of the Use of Sodium Bicarbonate for the Treatment of QRS Widening |pdf=|usr=}}{{26159649}} gab.com Text A Literature Review of the Use of Sodium Bicarbonate for the Treatment of QRS Widening JO: J Med Toxicol http://www.kidney.de/pget.php?&tw=1&pmid=26159649 #FOAvip Sodium bicarbonate is a well-known antidote for tricyclic antidepressant (TCA) poisoning. It has been used for over half a century to treat toxin-induced sodium channel blockade as evidenced by QRS widening on the electrocardiogram (ECG). The purpose of this review is to describe the literature regarding electrophysiological mechanisms and clinical use of this antidote after poisoning by tricyclic antidepressants and other agents. This article will also address the literature supporting an increased serum sodium concentration, alkalemia, or the combination of both as the responsible mechanism(s) for sodium bicarbonate's antidotal properties. While sodium bicarbonate has been used as a treatment for cardiac sodium channel blockade for multiple other agents including citalopram, cocaine, flecainide, diphenhydramine, propoxyphene, and lamotrigine, it has uncertain efficacy with bupropion, propranolol, and taxine-containing plants. Twit Text FOAVip A Literature Review of the Use of Sodium Bicarbonate for the Treatment of QRS Widening ????J Med Toxicol http://www.kidney.de/pget.php?&tw=1&pmid=26159649 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26159649 #FOAvip English Wikipedia <ref>{{Cite pmid|26159649}}</ref> A Literature Review of the Use of Sodium Bicarbonate for the Treatment of QRS Widening #MMPMID26159649 Bruccoleri RE; Burns MM J Med Toxicol 2016[Mar]; 12 (1): 121-9 PMID26159649show ga Sodium bicarbonate is a well-known antidote for tricyclic antidepressant (TCA) poisoning. It has been used for over half a century to treat toxin-induced sodium channel blockade as evidenced by QRS widening on the electrocardiogram (ECG). The purpose of this review is to describe the literature regarding electrophysiological mechanisms and clinical use of this antidote after poisoning by tricyclic antidepressants and other agents. This article will also address the literature supporting an increased serum sodium concentration, alkalemia, or the combination of both as the responsible mechanism(s) for sodium bicarbonate's antidotal properties. While sodium bicarbonate has been used as a treatment for cardiac sodium channel blockade for multiple other agents including citalopram, cocaine, flecainide, diphenhydramine, propoxyphene, and lamotrigine, it has uncertain efficacy with bupropion, propranolol, and taxine-containing plants. |Action Potentials[MESH] |Anti-Arrhythmia Agents/poisoning/*therapeutic use[MESH] |Antidepressive Agents, Tricyclic/poisoning[MESH] |Antidotes/adverse effects/*therapeutic use[MESH] |Antimalarials/poisoning[MESH] |Arrhythmias, Cardiac/chemically induced/diagnosis/*drug therapy/physiopathology[MESH] |Drug Overdose/diagnosis/*drug therapy/etiology/physiopathology[MESH] |Heart Conduction System/*drug effects/physiopathology[MESH] |Heart Rate/*drug effects[MESH] |Humans[MESH] |Risk Factors[MESH] |Sodium Bicarbonate/adverse effects/*therapeutic use[MESH]A Literature Review of the Use of Sodium Bicarbonate for the Treatment(epmc).pdf DeepDyve Pubget Overpricing