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10.1089/jir.2014.0237 http://scihub22266oqcxt.onion/10.1089/jir.2014.0237 26134384!ä!26134384 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26134384&cmd=llinks): Failed to open stream: HTTP request failed! 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Hyporeactivity of Alveolar Macrophages and Higher Respiratory Cell Permissivity Characterize DBA/2J Mice Infected by Influenza A Virus |pdf=|usr=}}{{26134384}} gab.com Text Hyporeactivity of Alveolar Macrophages and Higher Respiratory Cell Permissivity Characterize DBA/2J Mice Infected by Influenza A Virus JO: J Interferon Cytokine Res http://www.kidney.de/pget.php?&tw=1&pmid=26134384 #FOAvip Influenza A virus remains a major public health problem. Mouse models have been widely used to study influenza infection in mammals. DBA/2J and C57BL/6J represent extremes in terms of susceptibility to influenza A infection among inbred laboratory mouse strains. Several studies focused specifically on the factors responsible for the susceptibility of DBA/2J or the resistance of C57BL/6J and resulted in impressive lists of candidate genes or factors over- or underexpressed in one of the strains. We adopted a different phenotypical approach to identify the critical steps of the infection process accounting for the differences between DBA/2J and C57BL/6J strains. We concluded that both a dysfunction of alveolar macrophages and an increased permissivity of respiratory cells rendered DBA/2J more susceptible to influenza infection. Twit Text FOAVip Hyporeactivity of Alveolar Macrophages and Higher Respiratory Cell Permissivity Characterize DBA/2J Mice Infected by Influenza A Virus ????J Interferon Cytokine Res http://www.kidney.de/pget.php?&tw=1&pmid=26134384 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26134384 #FOAvip English Wikipedia <ref>{{Cite pmid|26134384}}</ref> Hyporeactivity of Alveolar Macrophages and Higher Respiratory Cell Permissivity Characterize DBA/2J Mice Infected by Influenza A Virus #MMPMID26134384 Casanova T; Van de Paar E; Desmecht D; Garigliany MM J Interferon Cytokine Res 2015[Oct]; 35 (10): 808-20 PMID26134384show ga Influenza A virus remains a major public health problem. Mouse models have been widely used to study influenza infection in mammals. DBA/2J and C57BL/6J represent extremes in terms of susceptibility to influenza A infection among inbred laboratory mouse strains. Several studies focused specifically on the factors responsible for the susceptibility of DBA/2J or the resistance of C57BL/6J and resulted in impressive lists of candidate genes or factors over- or underexpressed in one of the strains. We adopted a different phenotypical approach to identify the critical steps of the infection process accounting for the differences between DBA/2J and C57BL/6J strains. We concluded that both a dysfunction of alveolar macrophages and an increased permissivity of respiratory cells rendered DBA/2J more susceptible to influenza infection. |Alveolar Epithelial Cells/*immunology/metabolism/virology[MESH] |Animals[MESH] |Chemokines/genetics/metabolism[MESH] |Complement System Proteins/immunology[MESH] |Cytokines/genetics/metabolism[MESH] |Disease Models, Animal[MESH] |Disease Resistance[MESH] |Disease Susceptibility[MESH] |Gene Expression[MESH] |Immunohistochemistry[MESH] |Influenza A Virus, H1N1 Subtype/immunology[MESH] |Influenza A virus/*immunology[MESH] |Lung/immunology/metabolism/pathology/virology[MESH] |Macrophages, Alveolar/*immunology/metabolism/virology[MESH] |Mice[MESH] |Mice, Inbred DBA[MESH] |N-Acetylneuraminic Acid/metabolism[MESH] |Orthomyxoviridae Infections/*immunology/mortality/pathology/virology[MESH] |Respiratory Mucosa/immunology/metabolism/pathology/virology[MESH]Hyporeactivity of Alveolar Macrophages and Higher Respiratory Cell Per(epmc).pdf DeepDyve Pubget Overpricing