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10.1016/j.semnephrol.2015.01.010 http://scihub22266oqcxt.onion/10.1016/j.semnephrol.2015.01.010 25795503!4476528!25795503     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25795503&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Semin+Nephrol 2015 ; 35 (1): 96-107 Nephropedia Template TP {{tp|p=25795503|t=2015. Renal endothelial injury and microvascular dysfunction in acute kidney injury |pdf=|usr=}}{{25795503}} gab.com Text Renal endothelial injury and microvascular dysfunction in acute kidney injury JO: Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=25795503 #FOAvip The kidney is comprised of heterogeneous cell populations that function together to perform a number of tightly controlled, complex and interdependent processes. Renal endothelial cells contribute to vascular tone, regulation of blood flow to local tissue beds, modulation of coagulation and inflammation, and vascular permeability. Both ischemia and sepsis have profound effects on the renal endothelium, resulting in microvascular dysregulation resulting in continued ischemia and further injury. In recent years, the concept of the vascular endothelium as an organ that is both the source of and target for inflammatory injury has become widely appreciated. Here we revisit the renal endothelium in the light of ever evolving molecular advances. Twit Text FOAVip Renal endothelial injury and microvascular dysfunction in acute kidney injury ????Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=25795503 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25795503 #FOAvip English Wikipedia <ref>{{Cite pmid|25795503}}</ref> Renal endothelial injury and microvascular dysfunction in acute kidney injury #MMPMID25795503 Verma SK; Molitoris BA Semin Nephrol 2015[Jan]; 35 (1): 96-107 PMID25795503show ga The kidney is comprised of heterogeneous cell populations that function together to perform a number of tightly controlled, complex and interdependent processes. Renal endothelial cells contribute to vascular tone, regulation of blood flow to local tissue beds, modulation of coagulation and inflammation, and vascular permeability. Both ischemia and sepsis have profound effects on the renal endothelium, resulting in microvascular dysregulation resulting in continued ischemia and further injury. In recent years, the concept of the vascular endothelium as an organ that is both the source of and target for inflammatory injury has become widely appreciated. Here we revisit the renal endothelium in the light of ever evolving molecular advances. |*Renal Circulation[MESH] |Acute Kidney Injury/etiology/metabolism/*physiopathology[MESH] |Endothelial Cells/metabolism/physiology[MESH] |Endothelium, Vascular/metabolism/*physiopathology[MESH] |Humans[MESH] |Ischemia/metabolism/*physiopathology[MESH] |Kidney/blood supply/metabolism/*physiopathology[MESH] |Microvessels/metabolism/*physiopathology[MESH]Renal endothelial injury and microvascular dysfunction in acute kidney(epmc).pdf DeepDyve Pubget Overpricing