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10.1038/aps.2013.89 http://scihub22266oqcxt.onion/10.1038/aps.2013.89 23892271/?report=reader!3764334!23892271     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Acta+Pharmacol+Sin 2013 ; 34 (9): 1121-30 Nephropedia Template TP {{tp|p=23892271|t=2013. Inflammatory responses in hypoxic ischemic encephalopathy |pdf=|usr=}}{{23892271}} gab.com Text Inflammatory responses in hypoxic ischemic encephalopathy JO: Acta Pharmacol Sin http://www.kidney.de/pget.php?&tw=1&pmid=23892271 #FOAvip Inflammation plays a critical role in mediating brain injury induced by neonatal hypoxic ischemic encephalopathy (HIE). The mechanisms underlying inflammatory responses to ischemia may be shared by neonatal and adult brains; however, HIE exhibits a unique inflammation phenotype that results from the immaturity of the neonatal immune system. This review will discuss the current knowledge concerning systemic and local inflammatory responses in the acute and subacute stages of HIE. The key components of inflammation, including immune cells, adhesion molecules, cytokines, chemokines and oxidative stress, will be reviewed, and the differences between neonatal and adult inflammatory responses to cerebral ischemic injury will also be discussed. Twit Text FOAVip Inflammatory responses in hypoxic ischemic encephalopathy ????Acta Pharmacol Sin http://www.kidney.de/pget.php?&tw=1&pmid=23892271 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23892271 #FOAvip English Wikipedia <ref>{{Cite pmid|23892271}}</ref> Inflammatory responses in hypoxic ischemic encephalopathy #MMPMID23892271 Liu F; McCullough LD Acta Pharmacol Sin 2013[Sep]; 34 (9): 1121-30 PMID23892271show ga Inflammation plays a critical role in mediating brain injury induced by neonatal hypoxic ischemic encephalopathy (HIE). The mechanisms underlying inflammatory responses to ischemia may be shared by neonatal and adult brains; however, HIE exhibits a unique inflammation phenotype that results from the immaturity of the neonatal immune system. This review will discuss the current knowledge concerning systemic and local inflammatory responses in the acute and subacute stages of HIE. The key components of inflammation, including immune cells, adhesion molecules, cytokines, chemokines and oxidative stress, will be reviewed, and the differences between neonatal and adult inflammatory responses to cerebral ischemic injury will also be discussed. |Animals[MESH] |Brain/metabolism/*pathology[MESH] |Cytokines/immunology/metabolism[MESH] |Humans[MESH] |Hypoxia-Ischemia, Brain/metabolism/*pathology[MESH] |Macrophages/metabolism/pathology[MESH]Inflammatory responses in hypoxic ischemic encephalopathy (epmc).pdf DeepDyve Pubget Overpricing