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10.1152/ajpendo.1990.258.4.E555 http://scihub22266oqcxt.onion/10.1152/ajpendo.1990.258.4.E555 2333957!ä!2333957 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Physiol 1990 ; 258 (4 Pt 1): E555-61 Nephropedia Template TP {{tp|p=2333957|t=1990. Sustained stimulation of aldosterone production by angiotensin II is potentiated by nickel |pdf=|usr=}}{{2333957}} gab.com Text Sustained stimulation of aldosterone production by angiotensin II is potentiated by nickel JO: Am J Physiol http://www.kidney.de/pget.php?&tw=1&pmid=2333957 #FOAvip Angiotensin-induced aldosterone production by superfused adrenal glomerulosa cells was potentiated by Ni2+ (0.1 mM), added either at the onset of stimulation with angiotensin II or 1 h later. Nickel did not influence the effect of adrenocorticotropic hormone or potassium on aldosterone production. Nickel failed to modify angiotensin-induced changes in phospholipid metabolism or the formation of inositol phosphates and slightly reduced the enhancement of 45Ca influx. Uptake of Ni2+ into glomerulosa cells was increased by depolarization in a dihydropyridine-insensitive manner. Because nickel selectively potentiates the sustained phase of the response to a calcium-mobilizing hormone, it may serve as a suitable tool in elucidating the signal transduction process during the sustained phase of stimulation. Twit Text FOAVip Sustained stimulation of aldosterone production by angiotensin II is potentiated by nickel ????Am J Physiol http://www.kidney.de/pget.php?&tw=1&pmid=2333957 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=2333957 #FOAvip English Wikipedia <ref>{{Cite pmid|2333957}}</ref> Sustained stimulation of aldosterone production by angiotensin II is potentiated by nickel #MMPMID2333957 Spat A; Balla I; Balla T; Enyedi P; Hajnoczky G; Rohacs T Am J Physiol 1990[Apr]; 258 (4 Pt 1): E555-61 PMID2333957show ga Angiotensin-induced aldosterone production by superfused adrenal glomerulosa cells was potentiated by Ni2+ (0.1 mM), added either at the onset of stimulation with angiotensin II or 1 h later. Nickel did not influence the effect of adrenocorticotropic hormone or potassium on aldosterone production. Nickel failed to modify angiotensin-induced changes in phospholipid metabolism or the formation of inositol phosphates and slightly reduced the enhancement of 45Ca influx. Uptake of Ni2+ into glomerulosa cells was increased by depolarization in a dihydropyridine-insensitive manner. Because nickel selectively potentiates the sustained phase of the response to a calcium-mobilizing hormone, it may serve as a suitable tool in elucidating the signal transduction process during the sustained phase of stimulation. |Aldosterone/*biosynthesis[MESH] |Angiotensin II/*pharmacology[MESH] |Animals[MESH] |Biological Transport, Active/drug effects[MESH] |Calcium/metabolism[MESH] |Cobalt/pharmacology[MESH] |Female[MESH] |In Vitro Techniques[MESH] |Inositol Phosphates/metabolism[MESH] |Kinetics[MESH] |Male[MESH] |Nickel/metabolism/*pharmacology[MESH] |Rats[MESH] |Rats, Inbred Strains[MESH]Sustained stimulation of aldosterone production by angiotensin II is p(epmc).pdf DeepDyve Pubget Overpricing