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10.1038/ki.2012.371 http://scihub22266oqcxt.onion/10.1038/ki.2012.371 23271485!ä!23271485 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Kidney+Int 2013 ; 83 (1): 16-8 Nephropedia Template TP {{tp|p=23271485|t=2013. Complement is crucial in the pathogenesis of ANCA-associated vasculitis |pdf=|usr=}}{{23271485}} gab.com Text Complement is crucial in the pathogenesis of ANCA-associated vasculitis JO: Kidney Int http://www.kidney.de/pget.php?&tw=1&pmid=23271485 #FOAvip Renal lesions in ANCA-associated vasculitis (AAV) show an absence or paucity of immune deposits. Therefore, complement was not considered a major pathogenic factor. Data from an animal model of AAV, however, suggest involvement of the alternative pathway of complement. The paper by Gou et al. demonstrates activation also of the alternative and final common pathways in patients with AAV. Thus, the complement system might be a target for treatment in human AAV. Twit Text FOAVip Complement is crucial in the pathogenesis of ANCA-associated vasculitis ????Kidney Int http://www.kidney.de/pget.php?&tw=1&pmid=23271485 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23271485 #FOAvip English Wikipedia <ref>{{Cite pmid|23271485}}</ref> Complement is crucial in the pathogenesis of ANCA-associated vasculitis #MMPMID23271485 Kallenberg CG; Heeringa P Kidney Int 2013[Jan]; 83 (1): 16-8 PMID23271485show ga Renal lesions in ANCA-associated vasculitis (AAV) show an absence or paucity of immune deposits. Therefore, complement was not considered a major pathogenic factor. Data from an animal model of AAV, however, suggest involvement of the alternative pathway of complement. The paper by Gou et al. demonstrates activation also of the alternative and final common pathways in patients with AAV. Thus, the complement system might be a target for treatment in human AAV. |*Complement Activation[MESH] |*Severity of Illness Index[MESH] |Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/*blood/*diagnosis[MESH] |Complement Factor B/*metabolism[MESH] |Female[MESH] |Humans[MESH]Complement is crucial in the pathogenesis of ANCA-associated vasculiti(epmc).pdf DeepDyve Pubget Overpricing