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10.1053/j.ajkd.2012.07.033 http://scihub22266oqcxt.onion/10.1053/j.ajkd.2012.07.033 23201160!ä!23201160 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Kidney+Dis 2013 ; 62 (2): 377-83 Nephropedia Template TP {{tp|p=23201160|t=2013. Evaluation of hypomagnesemia: lessons from disorders of tubular transport |pdf=|usr=}}{{23201160}} gab.com Text Evaluation of hypomagnesemia: lessons from disorders of tubular transport JO: Am J Kidney Dis http://www.kidney.de/pget.php?&tw=1&pmid=23201160 #FOAvip Hypomagnesemia is a highly prevalent clinical condition affecting a large number of hospitalized patients. A decrease in systemic magnesium concentration may lead to impaired function of both neurologic and cardiovascular systems. The kidney has a pivotal role in magnesium handling by adjusting the urinary excretion of this ion in order to maintain systemic concentrations within a narrow range. As such, the cause of hypomagnesemia can be related to increments in the renal excretion of this cation. Many hypomagnesemic disorders also have characteristic changes in the renal reabsorptive capacity for other electrolytes, leading to symptoms that sometimes obscure the clinical presentation. For instance, changes in serum calcium concentration or its urinary excretion can aid in determining the underlying cause. Moreover, hypokalemia due to renal potassium losses often is associated with hypomagnesemia. Genetic defects in pathways controlling renal electrolyte transport impose the hypomagnesemic condition by facilitating renal losses. The discovery of the causative genes has greatly increased our understanding of how magnesium is transported by the kidney. Such knowledge is integral for the continued improvement of patient care with respect to bettering therapies and diagnosis. Twit Text FOAVip Evaluation of hypomagnesemia: lessons from disorders of tubular transport ????Am J Kidney Dis http://www.kidney.de/pget.php?&tw=1&pmid=23201160 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23201160 #FOAvip English Wikipedia <ref>{{Cite pmid|23201160}}</ref> Evaluation of hypomagnesemia: lessons from disorders of tubular transport #MMPMID23201160 Dimke H; Monnens L; Hoenderop JG; Bindels RJ Am J Kidney Dis 2013[Aug]; 62 (2): 377-83 PMID23201160show ga Hypomagnesemia is a highly prevalent clinical condition affecting a large number of hospitalized patients. A decrease in systemic magnesium concentration may lead to impaired function of both neurologic and cardiovascular systems. The kidney has a pivotal role in magnesium handling by adjusting the urinary excretion of this ion in order to maintain systemic concentrations within a narrow range. As such, the cause of hypomagnesemia can be related to increments in the renal excretion of this cation. Many hypomagnesemic disorders also have characteristic changes in the renal reabsorptive capacity for other electrolytes, leading to symptoms that sometimes obscure the clinical presentation. For instance, changes in serum calcium concentration or its urinary excretion can aid in determining the underlying cause. Moreover, hypokalemia due to renal potassium losses often is associated with hypomagnesemia. Genetic defects in pathways controlling renal electrolyte transport impose the hypomagnesemic condition by facilitating renal losses. The discovery of the causative genes has greatly increased our understanding of how magnesium is transported by the kidney. Such knowledge is integral for the continued improvement of patient care with respect to bettering therapies and diagnosis. |Adolescent[MESH] |Female[MESH] |Humans[MESH] |Magnesium/*blood[MESH]Evaluation of hypomagnesemia: lessons from disorders of tubular transp(epmc).pdf DeepDyve Pubget Overpricing