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10.1155/2012/608397 http://scihub22266oqcxt.onion/10.1155/2012/608397 22567283!3332212!22567283     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Nephrol 2012 ; 2012 (ä): 608397 Nephropedia Template TP {{tp|p=22567283|t=2012. Inflammation and oxidative stress in obesity-related glomerulopathy |pdf=|usr=}}{{22567283}} gab.com Text Inflammation and oxidative stress in obesity-related glomerulopathy JO: Int J Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=22567283 #FOAvip Obesity-related glomerulopathy is an increasing cause of end-stage renal disease. Obesity has been considered a state of chronic low-grade systemic inflammation and chronic oxidative stress. Augmented inflammation in adipose and kidney tissues promotes the progression of kidney damage in obesity. Adipose tissue, which is accumulated in obesity, is a key endocrine organ that produces multiple biologically active molecules, including leptin, adiponectin, resistin, that affect inflammation, and subsequent deregulation of cell function in renal glomeruli that leads to pathological changes. Oxidative stress is also associated with obesity-related renal diseases and may trigger the initiation or progression of renal damage in obesity. In this paper, we focus on inflammation and oxidative stress in the progression of obesity-related glomerulopathy and possible interventions to prevent kidney injury in obesity. Twit Text FOAVip Inflammation and oxidative stress in obesity-related glomerulopathy ????Int J Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=22567283 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22567283 #FOAvip English Wikipedia <ref>{{Cite pmid|22567283}}</ref> Inflammation and oxidative stress in obesity-related glomerulopathy #MMPMID22567283 Tang J; Yan H; Zhuang S Int J Nephrol 2012[]; 2012 (ä): 608397 PMID22567283show ga Obesity-related glomerulopathy is an increasing cause of end-stage renal disease. Obesity has been considered a state of chronic low-grade systemic inflammation and chronic oxidative stress. Augmented inflammation in adipose and kidney tissues promotes the progression of kidney damage in obesity. Adipose tissue, which is accumulated in obesity, is a key endocrine organ that produces multiple biologically active molecules, including leptin, adiponectin, resistin, that affect inflammation, and subsequent deregulation of cell function in renal glomeruli that leads to pathological changes. Oxidative stress is also associated with obesity-related renal diseases and may trigger the initiation or progression of renal damage in obesity. In this paper, we focus on inflammation and oxidative stress in the progression of obesity-related glomerulopathy and possible interventions to prevent kidney injury in obesity. äInflammation and oxidative stress in obesity-related glomerulopathy (epmc).pdf DeepDyve Pubget Overpricing