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10.1155/2012/381320 http://scihub22266oqcxt.onion/10.1155/2012/381320 22518311!3299271!22518311     free     free     free       Int+J+Nephrol 2012 ; 2012 (?): 381320 Nephropedia Template TP {{tp|p=22518311|t=2012. Reactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis and Renal Proximal Tubular Sodium Handling |pdf=|usr=}}{{22518311}} gab.com Text Reactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis and Renal Proximal Tubular Sodium Handling JO: Int J Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=22518311 #FOAvip The Na/K-ATPase is the primary force regulating renal sodium handling and plays a key role in both ion homeostasis and blood pressure regulation. Recently, cardiotonic steroids (CTS)-mediated Na/K-ATPase signaling has been shown to regulate fibrosis, renal proximal tubule (RPT) sodium reabsorption, and experimental Dahl salt-sensitive hypertension in response to a high-salt diet. Reactive oxygen species (ROS) are an important modulator of nephron ion transport. As there is limited knowledge regarding the role of ROS-mediated fibrosis and RPT sodium reabsorption through the Na/K-ATPase, the focus of this review is to examine the possible role of ROS in the regulation of Na/K-ATPase activity, its signaling, fibrosis, and RPT sodium reabsorption. Twit Text FOAVip Reactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis and Renal Proximal Tubular Sodium Handling ????Int J Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=22518311 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22518311 #FOAvip English Wikipedia <ref>{{Cite pmid|22518311}}</ref> Reactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis and Renal Proximal Tubular Sodium Handling #MMPMID22518311 Liu J; Kennedy DJ; Yan Y; Shapiro JI Int J Nephrol 2012[]; 2012 (?): 381320 PMID22518311show ga The Na/K-ATPase is the primary force regulating renal sodium handling and plays a key role in both ion homeostasis and blood pressure regulation. Recently, cardiotonic steroids (CTS)-mediated Na/K-ATPase signaling has been shown to regulate fibrosis, renal proximal tubule (RPT) sodium reabsorption, and experimental Dahl salt-sensitive hypertension in response to a high-salt diet. Reactive oxygen species (ROS) are an important modulator of nephron ion transport. As there is limited knowledge regarding the role of ROS-mediated fibrosis and RPT sodium reabsorption through the Na/K-ATPase, the focus of this review is to examine the possible role of ROS in the regulation of Na/K-ATPase activity, its signaling, fibrosis, and RPT sodium reabsorption. ?Reactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis a(epmc).pdf DeepDyve Pubget Overpricing