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10.3389/fphys.2012.00083 http://scihub22266oqcxt.onion/10.3389/fphys.2012.00083 22509167!3321473!22509167     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Physiol 2012 ; 3 (ä): 83 Nephropedia Template TP {{tp|p=22509167|t=2012. Gasotransmitters: novel regulators of epithelial na(+) transport?|pdf=|usr=}}{{22509167}} gab.com Text Gasotransmitters: novel regulators of epithelial na(+) transport JO: Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=22509167 #FOAvip The vectorial transport of Na(+) across epithelia is crucial for the maintenance of Na(+) and water homeostasis in organs such as the kidneys, lung, or intestine. Dysregulated Na(+) transport processes are associated with various human diseases such as hypertension, the salt-wasting syndrome pseudohypoaldosteronism type 1, pulmonary edema, cystic fibrosis, or intestinal disorders, which indicate that a precise regulation of epithelial Na(+) transport is essential. Novel regulatory signaling molecules are gasotransmitters. There are currently three known gasotransmitters: nitric oxide (NO), carbon monoxide (CO), and hydrogen sulfide (H(2)S). These molecules are endogenously produced in mammalian cells by specific enzymes and have been shown to regulate various physiological processes. There is a growing body of evidence which indicates that gasotransmitters may also regulate Na(+) transport across epithelia. This review will summarize the available data concerning NO, CO, and H(2)S dependent regulation of epithelial Na(+) transport processes and will discuss whether or not these mediators can be considered as true physiological regulators of epithelial Na(+) transport biology. Twit Text FOAVip Gasotransmitters: novel regulators of epithelial na(+) transport ????Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=22509167 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22509167 #FOAvip English Wikipedia <ref>{{Cite pmid|22509167}}</ref> Gasotransmitters: novel regulators of epithelial na(+) transport? #MMPMID22509167 Althaus M Front Physiol 2012[]; 3 (ä): 83 PMID22509167show ga The vectorial transport of Na(+) across epithelia is crucial for the maintenance of Na(+) and water homeostasis in organs such as the kidneys, lung, or intestine. Dysregulated Na(+) transport processes are associated with various human diseases such as hypertension, the salt-wasting syndrome pseudohypoaldosteronism type 1, pulmonary edema, cystic fibrosis, or intestinal disorders, which indicate that a precise regulation of epithelial Na(+) transport is essential. Novel regulatory signaling molecules are gasotransmitters. There are currently three known gasotransmitters: nitric oxide (NO), carbon monoxide (CO), and hydrogen sulfide (H(2)S). These molecules are endogenously produced in mammalian cells by specific enzymes and have been shown to regulate various physiological processes. There is a growing body of evidence which indicates that gasotransmitters may also regulate Na(+) transport across epithelia. This review will summarize the available data concerning NO, CO, and H(2)S dependent regulation of epithelial Na(+) transport processes and will discuss whether or not these mediators can be considered as true physiological regulators of epithelial Na(+) transport biology. äGasotransmitters: novel regulators of epithelial na(+) transport?(epmc).pdf DeepDyve Pubget Overpricing