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10.1042/CS20110506

http://scihub22266oqcxt.onion/10.1042/CS20110506
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22248353!ä!22248353

suck abstract from ncbi


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pmid22248353      Clin+Sci+(Lond) 2012 ; 122 (9): 397-407
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  • Endothelial cells and magnesium: implications in atherosclerosis #MMPMID22248353
  • Maier JA
  • Clin Sci (Lond) 2012[May]; 122 (9): 397-407 PMID22248353show ga
  • There is no doubt that the functional and structural integrity of the endothelium is critical in maintaining vascular homoeostasis and in preventing atherosclerosis. In the light of epidemiological and experimental studies, magnesium deficiency is emerging as an inducer of endothelial dysfunction. In particular, data on the effects of low extracellular magnesium on cultured endothelial cells reinforce the idea that correcting magnesium homoeostasis might be a helpful and inexpensive intervention to prevent and treat endothelial dysfunction and, consequently, atherosclerosis.
  • |Animals[MESH]
  • |Atherosclerosis/drug therapy/*etiology/*physiopathology[MESH]
  • |Capillary Permeability[MESH]
  • |Endothelial Cells/drug effects/*physiology[MESH]
  • |Humans[MESH]
  • |Interleukin-1alpha/physiology[MESH]
  • |Magnesium Deficiency/*complications/drug therapy/physiopathology[MESH]
  • |Magnesium/pharmacology/*physiology[MESH]
  • |Models, Cardiovascular[MESH]
  • |NF-kappa B/physiology[MESH]
  • |Oxidative Stress[MESH]
  • |Peptide Hydrolases/biosynthesis[MESH]


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