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10.1016/j.antiviral.2011.07.011 http://scihub22266oqcxt.onion/10.1016/j.antiviral.2011.07.011 21798291!ä!21798291 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Antiviral+Res 2011 ; 91 (3): 330-4 Nephropedia Template TP {{tp|p=21798291|t=2011. Antiviral effect of a selective COX-2 inhibitor on H5N1 infection in vitro |pdf=|usr=}}{{21798291}} gab.com Text Antiviral effect of a selective COX-2 inhibitor on H5N1 infection in vitro JO: Antiviral Res http://www.kidney.de/pget.php?&tw=1&pmid=21798291 #FOAvip A selective cyclooxygenase-2 (COX-2) inhibitor has been previously shown to suppress the hyper-induced pro-inflammatory responses in H5N1 infected primary human cells. Here, we demonstrate that COX-2 inhibitors suppress H5N1 virus replication in human macrophages suggesting that H5N1 virus replication (more so than seasonal H1N1 virus) is dependent on activation of COX-2 dependent signaling pathways in host cells. COX-2 and its downstream signaling pathways deserve detailed investigation as a novel therapeutic target for treatment of H5N1 disease. Twit Text FOAVip Antiviral effect of a selective COX-2 inhibitor on H5N1 infection in vitro ????Antiviral Res http://www.kidney.de/pget.php?&tw=1&pmid=21798291 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=21798291 #FOAvip English Wikipedia <ref>{{Cite pmid|21798291}}</ref> Antiviral effect of a selective COX-2 inhibitor on H5N1 infection in vitro #MMPMID21798291 Lee SM; Gai WW; Cheung TK; Peiris JS Antiviral Res 2011[Sep]; 91 (3): 330-4 PMID21798291show ga A selective cyclooxygenase-2 (COX-2) inhibitor has been previously shown to suppress the hyper-induced pro-inflammatory responses in H5N1 infected primary human cells. Here, we demonstrate that COX-2 inhibitors suppress H5N1 virus replication in human macrophages suggesting that H5N1 virus replication (more so than seasonal H1N1 virus) is dependent on activation of COX-2 dependent signaling pathways in host cells. COX-2 and its downstream signaling pathways deserve detailed investigation as a novel therapeutic target for treatment of H5N1 disease. |Animals[MESH] |Antiviral Agents/pharmacology[MESH] |Birds[MESH] |Cells, Cultured[MESH] |Cyclooxygenase 2 Inhibitors/pharmacology[MESH] |Cyclooxygenase 2/genetics/*metabolism[MESH] |Dose-Response Relationship, Drug[MESH] |Humans[MESH] |Influenza A Virus, H1N1 Subtype/drug effects/physiology[MESH] |Influenza A Virus, H5N1 Subtype/*drug effects/physiology[MESH] |Influenza in Birds/drug therapy/virology[MESH] |Influenza, Human/*drug therapy/virology[MESH] |Macrophages/cytology/*drug effects/virology[MESH] |Reverse Transcriptase Polymerase Chain Reaction[MESH] |Signal Transduction/*drug effects[MESH] |Sulfonamides/*pharmacology[MESH] |Viral Proteins/genetics/*metabolism[MESH]Antiviral effect of a selective COX-2 inhibitor on H5N1 infection in v(epmc).pdf DeepDyve Pubget Overpricing