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10.4061/2010/316862

http://scihub22266oqcxt.onion/10.4061/2010/316862
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21052549!2968719!21052549
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suck abstract from ncbi

pmid21052549      Stroke+Res+Treat 2010 ; 2010 (?): 316862
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  • Modes of Neuronal Calcium Entry and Homeostasis following Cerebral Ischemia #MMPMID21052549
  • Cross JL; Meloni BP; Bakker AJ; Lee S; Knuckey NW
  • Stroke Res Treat 2010[Nov]; 2010 (?): 316862 PMID21052549show ga
  • One of the major instigators leading to neuronal cell death and brain damage following cerebral ischemia is calcium dysregulation. The neuron's inability to maintain calcium homeostasis is believed to be a result of increased calcium influx and impaired calcium extrusion across the plasma membrane. The need to better understand the cellular and biochemical mechanisms of calcium dysregulation contributing to neuronal loss following stroke/cerebral ischemia is essential for the development of new treatments in order to reduce ischemic brain injury. The aim of this paper is to provide a concise overview of the various calcium influx pathways in response to ischemia and how neuronal cells attempts to overcome this calcium overload.
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