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10.1002/jlb.50.2.160 http://scihub22266oqcxt.onion/10.1002/jlb.50.2.160 2072033!ä!2072033 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=2072033&cmd=llinks): Failed to open stream: HTTP request failed! 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The ability of polymorphonuclear leukocyte priming agents to overcome influenza A virus-induced cell dysfunction |pdf=|usr=}}{{2072033}} gab.com Text The ability of polymorphonuclear leukocyte priming agents to overcome influenza A virus-induced cell dysfunction JO: J Leukoc Biol http://www.kidney.de/pget.php?&tw=1&pmid=2072033 #FOAvip The major mortality and morbidity resulting from influenza virus infections are due to secondary bacterial infections which occur in association with virus-induced inhibition of polymorphonuclear leukocyte (PMNL) function. The present study was undertaken to determine if compounds which prime PMNL function to subsequent stimulation with N-formylmethionyl-leucylphenylalanine (FMLP) or phorbol 12-myristate 13-acetate (PMA) can overcome influenza A virus (IAV)-induced inhibition of the PMNL chemiluminescence response to these stimuli. Granulocyte-macrophage colony stimulating factor (GM-CSF), guanosine triphosphate (GTP), and 1-oleoyl-2-acetylglycerol (OAG) were able to prime the PMNL response to FMLP and/or PMA and totally or partially overcome IAV-induced PMNL dysfunction in cells stimulated with FMLP or PMA. A direct correlation was found between the extent of PMNL priming due to GM-CSF, GTP, and OAG and the capacity of these compounds to overcome virus-induced PMNL dysfunction. The implications of these findings in regard to the mechanism by which priming agents overcome IAV-induced cell dysfunction and the potential of these compounds as therapeutic agents to treat secondary bacterial infections are discussed. Twit Text FOAVip The ability of polymorphonuclear leukocyte priming agents to overcome influenza A virus-induced cell dysfunction ????J Leukoc Biol http://www.kidney.de/pget.php?&tw=1&pmid=2072033 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=2072033 #FOAvip English Wikipedia <ref>{{Cite pmid|2072033}}</ref> The ability of polymorphonuclear leukocyte priming agents to overcome influenza A virus-induced cell dysfunction #MMPMID2072033 Abramson JS; Wagner MP; Ralston EP; Wei Y; Wheeler JG J Leukoc Biol 1991[Aug]; 50 (2): 160-6 PMID2072033show ga The major mortality and morbidity resulting from influenza virus infections are due to secondary bacterial infections which occur in association with virus-induced inhibition of polymorphonuclear leukocyte (PMNL) function. The present study was undertaken to determine if compounds which prime PMNL function to subsequent stimulation with N-formylmethionyl-leucylphenylalanine (FMLP) or phorbol 12-myristate 13-acetate (PMA) can overcome influenza A virus (IAV)-induced inhibition of the PMNL chemiluminescence response to these stimuli. Granulocyte-macrophage colony stimulating factor (GM-CSF), guanosine triphosphate (GTP), and 1-oleoyl-2-acetylglycerol (OAG) were able to prime the PMNL response to FMLP and/or PMA and totally or partially overcome IAV-induced PMNL dysfunction in cells stimulated with FMLP or PMA. A direct correlation was found between the extent of PMNL priming due to GM-CSF, GTP, and OAG and the capacity of these compounds to overcome virus-induced PMNL dysfunction. The implications of these findings in regard to the mechanism by which priming agents overcome IAV-induced cell dysfunction and the potential of these compounds as therapeutic agents to treat secondary bacterial infections are discussed. |Diglycerides/pharmacology[MESH] |Granulocyte-Macrophage Colony-Stimulating Factor/pharmacology[MESH] |Guanosine Triphosphate/pharmacology[MESH] |Hemagglutinin Glycoproteins, Influenza Virus[MESH] |Hemagglutinins, Viral/*pharmacology[MESH] |Humans[MESH] |In Vitro Techniques[MESH] |Influenza A virus/*immunology[MESH] |Luminescent Measurements[MESH] |N-Formylmethionine Leucyl-Phenylalanine/*pharmacology[MESH] |Neutrophils/drug effects/microbiology/*physiology[MESH] |Recombinant Proteins/pharmacology[MESH] |Tetradecanoylphorbol Acetate/*pharmacology[MESH]The ability of polymorphonuclear leukocyte priming agents to overcome (epmc).pdf DeepDyve Pubget Overpricing