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10.1080/08998280.2010.11928628 http://scihub22266oqcxt.onion/10.1080/08998280.2010.11928628 20671821!2900977!20671821     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Proc+(Bayl+Univ+Med+Cent) 2010 ; 23 (3): 250-5 Nephropedia Template TP {{tp|p=20671821|t=2010. Causes and management of drug-induced long QT syndrome |pdf=|usr=}}{{20671821}} gab.com Text Causes and management of drug-induced long QT syndrome JO: Proc (Bayl Univ Med Cent) http://www.kidney.de/pget.php?&tw=1&pmid=20671821 #FOAvip Long QT syndrome (LQTS) is characterized by inherited or acquired prolonged QT interval on the surface electrocardiogram. This can lead to torsade de pointes ventricular tachycardia (TdP VT) and ventricular fibrillation. In the acquired form of the disease, medications from several classes can cause TdP VT or potentiate the electrocardiographic findings. These include class IA and III antiarrhythmics, antibiotics (macrolides and quinolones), antidepressants (tricyclics and selective serotonin reuptake inhibitors), antipsychotics (haloperidol and phenothiazines), and antiemetics (ondansetron and prochlorperazine). We present four cases of drug-induced LQTS resulting in life-threatening cardiac arrhythmias. Antiarrhythmic medications were the cause in two cases, and the other two cases involved noncardiac medications. All four patients had at least one risk factor for LQTS in addition to the offending drug, including female gender, hypokalemia, hypomagnesemia, and bradycardia. In one patient, amiodarone was administered for treatment of VT, although the correct diagnosis was actually TdP VT. In patients with polymorphic VT or ventricular fibrillation without a significant history of cardiovascular disease, drug-induced LQTS should be high in the differential diagnosis. Prompt diagnosis is key, as amiodarone, while often used to suppress VT, is potentially harmful in the setting of LQTS and TdP VT. Twit Text FOAVip Causes and management of drug-induced long QT syndrome ????Proc (Bayl Univ Med Cent) http://www.kidney.de/pget.php?&tw=1&pmid=20671821 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=20671821 #FOAvip English Wikipedia <ref>{{Cite pmid|20671821}}</ref> Causes and management of drug-induced long QT syndrome #MMPMID20671821 Ayad RF; Assar MD; Simpson L; Garner JB; Schussler JM Proc (Bayl Univ Med Cent) 2010[Jul]; 23 (3): 250-5 PMID20671821show ga Long QT syndrome (LQTS) is characterized by inherited or acquired prolonged QT interval on the surface electrocardiogram. This can lead to torsade de pointes ventricular tachycardia (TdP VT) and ventricular fibrillation. In the acquired form of the disease, medications from several classes can cause TdP VT or potentiate the electrocardiographic findings. These include class IA and III antiarrhythmics, antibiotics (macrolides and quinolones), antidepressants (tricyclics and selective serotonin reuptake inhibitors), antipsychotics (haloperidol and phenothiazines), and antiemetics (ondansetron and prochlorperazine). We present four cases of drug-induced LQTS resulting in life-threatening cardiac arrhythmias. Antiarrhythmic medications were the cause in two cases, and the other two cases involved noncardiac medications. All four patients had at least one risk factor for LQTS in addition to the offending drug, including female gender, hypokalemia, hypomagnesemia, and bradycardia. In one patient, amiodarone was administered for treatment of VT, although the correct diagnosis was actually TdP VT. In patients with polymorphic VT or ventricular fibrillation without a significant history of cardiovascular disease, drug-induced LQTS should be high in the differential diagnosis. Prompt diagnosis is key, as amiodarone, while often used to suppress VT, is potentially harmful in the setting of LQTS and TdP VT. äCauses and management of drug-induced long QT syndrome (epmc).pdf DeepDyve Pubget Overpricing