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10.1097/SMJ.0b013e3181e07751 http://scihub22266oqcxt.onion/10.1097/SMJ.0b013e3181e07751 20531067!ä!20531067 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\20531067.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       South+Med+J 2010 ; 103 (7): 654-61 Nephropedia Template TP {{tp|p=20531067|t=2010. Hepatorenal syndrome |pdf=|usr=}}{{20531067}} gab.com Text Hepatorenal syndrome JO: South Med J http://www.kidney.de/pget.php?&tw=1&pmid=20531067 #FOAvip Acute kidney injury (AKI) secondary to hepatorenal syndrome (HRS) is an ominous complication of end-stage liver disease (ESLD). In HRS, splanchnic and peripheral vasodilatation with reduction in effective arterial volume causes activation of mechanisms leading to intense renal vasoconstriction and functional AKI. HRS is a diagnosis of exclusion and all other causes of AKI (especially prerenal azotemia) have to be considered and excluded. Spontaneous bacterial peritonitis (SBP) frequently precipitates HRS and should be ruled out in all ESLD patients presenting with AKI. Prompt therapy of SBP with intravenous antibiotics and albumin lessens the risk of developing HRS. Combined use of intravenous albumin, splanchnic and/or peripheral vasoconstrictors, and renal replacement therapy (RRT) are only bridges to early liver transplantation (or combined liver-kidney transplantation in selected patients). Transplantation is the only definitive way of improving the long-term prognosis. Close collaboration between hospitalists/internists managing HRS patients and hepatology and nephrology consultants is critically important. Twit Text FOAVip Hepatorenal syndrome ????South Med J http://www.kidney.de/pget.php?&tw=1&pmid=20531067 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=20531067 #FOAvip English Wikipedia <ref>{{Cite pmid|20531067}}</ref> Hepatorenal syndrome #MMPMID20531067 Venkat D; Venkat KK South Med J 2010[Jul]; 103 (7): 654-61 PMID20531067show ga Acute kidney injury (AKI) secondary to hepatorenal syndrome (HRS) is an ominous complication of end-stage liver disease (ESLD). In HRS, splanchnic and peripheral vasodilatation with reduction in effective arterial volume causes activation of mechanisms leading to intense renal vasoconstriction and functional AKI. HRS is a diagnosis of exclusion and all other causes of AKI (especially prerenal azotemia) have to be considered and excluded. Spontaneous bacterial peritonitis (SBP) frequently precipitates HRS and should be ruled out in all ESLD patients presenting with AKI. Prompt therapy of SBP with intravenous antibiotics and albumin lessens the risk of developing HRS. Combined use of intravenous albumin, splanchnic and/or peripheral vasoconstrictors, and renal replacement therapy (RRT) are only bridges to early liver transplantation (or combined liver-kidney transplantation in selected patients). Transplantation is the only definitive way of improving the long-term prognosis. Close collaboration between hospitalists/internists managing HRS patients and hepatology and nephrology consultants is critically important. |*Hepatorenal Syndrome/diagnosis/epidemiology/etiology/prevention & control/therapy[MESH] |Anti-Bacterial Agents/therapeutic use[MESH] |Humans[MESH] |Incidence[MESH] |Liver Transplantation[MESH] |Prognosis[MESH] |Renal Replacement Therapy[MESH]Hepatorenal syndrome (epmc).pdf DeepDyve Pubget Overpricing