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10.1016/j.arcmed.2009.10.001 http://scihub22266oqcxt.onion/10.1016/j.arcmed.2009.10.001 20304252!ä!20304252 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Arch+Med+Res 2009 ; 40 (8): 655-61 Nephropedia Template TP {{tp|p=20304252|t=2009. Comparison of the pathology caused by H1N1, H5N1, and H3N2 influenza viruses |pdf=|usr=}}{{20304252}} gab.com Text Comparison of the pathology caused by H1N1, H5N1, and H3N2 influenza viruses JO: Arch Med Res http://www.kidney.de/pget.php?&tw=1&pmid=20304252 #FOAvip The spectrum of morbidity and mortality of H1N1, H5N1, and H3N2 influenza A viruses relates to the pathology they produce. In this review, we describe and compare the pathology of these viruses in human cases and animal models. The 1918 H1N1, the novel 2009 H1N1 pandemic virus, and H5N1 show inflammation, congestion, and epithelial necrosis of the larger airways (trachea, bronchi and bronchioles) with extension into the alveoli causing diffuse alveolar damage. Seasonal influenza A viruses (H3N2 and H1N1) have primarily caused inflammation, congestion and epithelial necrosis of the larger airways with lesser extension of the inflammatory process to alveoli. Localization of the inflammation and cellular damage relate to the presence of virus in different cell types. Infections with 1918 H1N1, the novel 2009 H1N1 pandemic virus, and H5N1 show virus in mucosal epithelial cells of the airways (from the nasopharynx to the bronchioles), alveolar macrophages, and pneumocytes, whereas infections with seasonal influenza viruses show viral antigens primarily in mucosal epithelial cells of the larger airways. The increased morbidity that has been encountered with the 2009 H1N1 virus is related to infection of cells in the upper and lower airways. The 2009 H1N1 virus shows similar pathology to that encountered with other highly virulent influenza A viruses such as the 1918 H1N1 and H5N1 viruses. Twit Text FOAVip Comparison of the pathology caused by H1N1, H5N1, and H3N2 influenza viruses ????Arch Med Res http://www.kidney.de/pget.php?&tw=1&pmid=20304252 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=20304252 #FOAvip English Wikipedia <ref>{{Cite pmid|20304252}}</ref> Comparison of the pathology caused by H1N1, H5N1, and H3N2 influenza viruses #MMPMID20304252 Guarner J; Falcon-Escobedo R Arch Med Res 2009[Nov]; 40 (8): 655-61 PMID20304252show ga The spectrum of morbidity and mortality of H1N1, H5N1, and H3N2 influenza A viruses relates to the pathology they produce. In this review, we describe and compare the pathology of these viruses in human cases and animal models. The 1918 H1N1, the novel 2009 H1N1 pandemic virus, and H5N1 show inflammation, congestion, and epithelial necrosis of the larger airways (trachea, bronchi and bronchioles) with extension into the alveoli causing diffuse alveolar damage. Seasonal influenza A viruses (H3N2 and H1N1) have primarily caused inflammation, congestion and epithelial necrosis of the larger airways with lesser extension of the inflammatory process to alveoli. Localization of the inflammation and cellular damage relate to the presence of virus in different cell types. Infections with 1918 H1N1, the novel 2009 H1N1 pandemic virus, and H5N1 show virus in mucosal epithelial cells of the airways (from the nasopharynx to the bronchioles), alveolar macrophages, and pneumocytes, whereas infections with seasonal influenza viruses show viral antigens primarily in mucosal epithelial cells of the larger airways. The increased morbidity that has been encountered with the 2009 H1N1 virus is related to infection of cells in the upper and lower airways. The 2009 H1N1 virus shows similar pathology to that encountered with other highly virulent influenza A viruses such as the 1918 H1N1 and H5N1 viruses. |*Lung/immunology/pathology/virology[MESH] |Alveolar Epithelial Cells/immunology/pathology/virology[MESH] |Animals[MESH] |Disease Models, Animal[MESH] |Disease Outbreaks[MESH] |Humans[MESH] |Influenza A Virus, H1N1 Subtype/*pathogenicity[MESH] |Influenza A Virus, H3N2 Subtype/*pathogenicity[MESH] |Influenza A Virus, H5N1 Subtype/*pathogenicity[MESH]Comparison of the pathology caused by H1N1, H5N1, and H3N2 influenza v(epmc).pdf DeepDyve Pubget Overpricing