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http://scihub22266oqcxt.onion/ 19377250!ä!19377250 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\19377250.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Iran+J+Kidney+Dis 2009 ; 3 (1): 1-6 Nephropedia Template TP {{tp|p=19377250|t=2009. New concepts in pathogenesis of renal hypophosphatemic syndromes |pdf=|usr=}}{{19377250}} gab.com Text New concepts in pathogenesis of renal hypophosphatemic syndromes JO: Iran J Kidney Dis http://www.kidney.de/pget.php?&tw=1&pmid=19377250 #FOAvip During the past decades, our knowledge of renal phosphate handling has advanced dramatically. This advance is primarily due to the discovery of sodium-phosphate transport channels and their regulation in health and disease. The discovery of phosphatonins, initially in patients with tumor-induced osteomalacia, has not only allowed us to develop a better understanding of several rare diseases including vitamin D-resistant rickets, but also it has expanded our knowledge of the dynamic interaction between the bone and the kidney critical to bone mineralization. In this review, the author focuses on these new developments and their importance to our understanding of phosphate homeostasis in health and disease. Twit Text FOAVip New concepts in pathogenesis of renal hypophosphatemic syndromes ????Iran J Kidney Dis http://www.kidney.de/pget.php?&tw=1&pmid=19377250 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=19377250 #FOAvip English Wikipedia <ref>{{Cite pmid|19377250}}</ref> New concepts in pathogenesis of renal hypophosphatemic syndromes #MMPMID19377250 Rastegar A Iran J Kidney Dis 2009[Jan]; 3 (1): 1-6 PMID19377250show ga During the past decades, our knowledge of renal phosphate handling has advanced dramatically. This advance is primarily due to the discovery of sodium-phosphate transport channels and their regulation in health and disease. The discovery of phosphatonins, initially in patients with tumor-induced osteomalacia, has not only allowed us to develop a better understanding of several rare diseases including vitamin D-resistant rickets, but also it has expanded our knowledge of the dynamic interaction between the bone and the kidney critical to bone mineralization. In this review, the author focuses on these new developments and their importance to our understanding of phosphate homeostasis in health and disease. |Fibroblast Growth Factor-23[MESH] |Fibroblast Growth Factors/metabolism[MESH] |Humans[MESH] |Hypophosphatemia/genetics/*metabolism[MESH] |Kidney/*metabolism[MESH] |Mutation[MESH] |Phosphates/metabolism[MESH] |Sodium-Phosphate Cotransporter Proteins/genetics[MESH] |Syndrome[MESH]New concepts in pathogenesis of renal hypophosphatemic syndromes (epmc).pdf DeepDyve Pubget Overpricing