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10.1080/15287399109531565 http://scihub22266oqcxt.onion/10.1080/15287399109531565 1920529!ä!1920529 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=1920529&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Toxicol+Environ+Health 1991 ; 34 (2): 259-73 Nephropedia Template TP {{tp|p=1920529|t=1991. Enhanced and prolonged pulmonary influenza virus infection following phosgene inhalation |pdf=|usr=}}{{1920529}} gab.com Text Enhanced and prolonged pulmonary influenza virus infection following phosgene inhalation JO: J Toxicol Environ Health http://www.kidney.de/pget.php?&tw=1&pmid=1920529 #FOAvip Animal infectivity models have been important in the demonstration of enhanced susceptibility to viral and bacterial infection as a result of low-level toxicant exposure. This study demonstrated an enhanced and prolonged viral infection using an influenza virus infectivity model in the rat following exposure to the toxicant gas phosgene. Fischer-344 rats exposed to either air or a sublethal concentration of phosgene demonstrated peak pulmonary influenza virus titers 1 d after infection. Virus titers in rats exposed to air declined rapidly falling below detectable levels by 4 d after infection. However, a significantly enhanced and prolonged pulmonary influenza virus infection was observed on d 3 and 4 after infection in rats exposed to phosgene. Virus was cleared below detectable limits on d 5 after infection in animals exposed to phosgene. Thus, inhalation of sublethal concentrations of phosgene resulted in an increased severity of pulmonary influenza virus infection. This study provides a demonstration of the effective use of a rat viral infectivity model to detect the immunotoxicity of inhaled pollutants. This model will allow future studies to focus on the immunological mechanism(s) responsible for the enhanced and prolonged pulmonary influenza virus infection. Twit Text FOAVip Enhanced and prolonged pulmonary influenza virus infection following phosgene inhalation ????J Toxicol Environ Health http://www.kidney.de/pget.php?&tw=1&pmid=1920529 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=1920529 #FOAvip English Wikipedia <ref>{{Cite pmid|1920529}}</ref> Enhanced and prolonged pulmonary influenza virus infection following phosgene inhalation #MMPMID1920529 Ehrlich JP; Burleson GR J Toxicol Environ Health 1991[Oct]; 34 (2): 259-73 PMID1920529show ga Animal infectivity models have been important in the demonstration of enhanced susceptibility to viral and bacterial infection as a result of low-level toxicant exposure. This study demonstrated an enhanced and prolonged viral infection using an influenza virus infectivity model in the rat following exposure to the toxicant gas phosgene. Fischer-344 rats exposed to either air or a sublethal concentration of phosgene demonstrated peak pulmonary influenza virus titers 1 d after infection. Virus titers in rats exposed to air declined rapidly falling below detectable levels by 4 d after infection. However, a significantly enhanced and prolonged pulmonary influenza virus infection was observed on d 3 and 4 after infection in rats exposed to phosgene. Virus was cleared below detectable limits on d 5 after infection in animals exposed to phosgene. Thus, inhalation of sublethal concentrations of phosgene resulted in an increased severity of pulmonary influenza virus infection. This study provides a demonstration of the effective use of a rat viral infectivity model to detect the immunotoxicity of inhaled pollutants. This model will allow future studies to focus on the immunological mechanism(s) responsible for the enhanced and prolonged pulmonary influenza virus infection. |Administration, Inhalation[MESH] |Animals[MESH] |Bronchoalveolar Lavage Fluid/chemistry/cytology[MESH] |Cell Count[MESH] |Cell Survival/drug effects[MESH] |Disease Models, Animal[MESH] |Disease Susceptibility[MESH] |Immune Tolerance/*drug effects[MESH] |Influenza A virus/*drug effects/physiology[MESH] |Lung/drug effects/immunology/microbiology[MESH] |Lymphocytes/drug effects[MESH] |Macrophages/drug effects[MESH] |Male[MESH] |Neutrophils/drug effects[MESH] |Orthomyxoviridae Infections/*immunology[MESH] |Phosgene/administration & dosage/*toxicity[MESH] |Rats[MESH] |Rats, Inbred F344[MESH] |Viral Plaque Assay[MESH]Enhanced and prolonged pulmonary influenza virus infection following p(epmc).pdf DeepDyve Pubget Overpricing