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10.3346/jkms.2009.24.S1.S161 http://scihub22266oqcxt.onion/10.3346/jkms.2009.24.S1.S161 19194547!2633179!19194547     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 265.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 265.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Korean+Med+Sci 2009 ; 24 Suppl (Suppl 1): S161-9 Nephropedia Template TP {{tp|p=19194547|t=2009. Effects of thiazide on the expression of TRPV5, calbindin-D28K, and sodium transporters in hypercalciuric rats |pdf=|usr=}}{{19194547}} gab.com Text Effects of thiazide on the expression of TRPV5, calbindin-D28K, and sodium transporters in hypercalciuric rats JO: J Korean Med Sci http://www.kidney.de/pget.php?&tw=1&pmid=19194547 #FOAvip TRPV5 is believed to play an important role in the regulation of urinary calcium excretion. We assessed the effects of hydrochlorothiazide (HCTZ) on the expression of TRPV5, calbindin-D(28K), and several sodium transporters in hypercalciuric rats. Sprague-Dawley rats were divided into 4 groups; control, HCTZ, high salt, and high salt with HCTZ group in experiment 1; control, HCTZ, high calcium (Ca), and high Ca with HCTZ group in experiment 2. To quantitate the expression of TRPV5, calbindin-D(28K), and sodium transporters, western blotting was performed. In both experiments, HCTZ significantly decreased urinary calcium excretion. TRPV5 protein abundance decreased in all hypercalciuric rats, and restored by HCTZ in both high salt with HCTZ and high Ca with HCTZ group. Calbindin-D(28K) protein abundance increased in the high salt and high salt with HCTZ groups, but did not differ among groups in experiment 2. Protein abundance of NHE3 and NKCC2 decreased in all hypercalciuric rats, and were restored by HCTZ in only high Ca-induced hypercalciuric rats. In summary, protein abundance of TRPV5, NHE3, and NKCC2 decreased in all hypercalciuric rats. The hypocalciuric effect of HCTZ is associated with increased protein abundance of TRPV5 in high salt or calcium diet-induced hypercalciuric rats. Twit Text FOAVip Effects of thiazide on the expression of TRPV5, calbindin-D28K, and sodium transporters in hypercalciuric rats ????J Korean Med Sci http://www.kidney.de/pget.php?&tw=1&pmid=19194547 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=19194547 #FOAvip English Wikipedia <ref>{{Cite pmid|19194547}}</ref> Effects of thiazide on the expression of TRPV5, calbindin-D28K, and sodium transporters in hypercalciuric rats #MMPMID19194547 Jang HR; Kim S; Heo NJ; Lee JH; Kim HS; Nielsen S; Jeon US; Oh YK; Na KY; Joo KW; Han JS J Korean Med Sci 2009[Jan]; 24 Suppl (Suppl 1): S161-9 PMID19194547show ga TRPV5 is believed to play an important role in the regulation of urinary calcium excretion. We assessed the effects of hydrochlorothiazide (HCTZ) on the expression of TRPV5, calbindin-D(28K), and several sodium transporters in hypercalciuric rats. Sprague-Dawley rats were divided into 4 groups; control, HCTZ, high salt, and high salt with HCTZ group in experiment 1; control, HCTZ, high calcium (Ca), and high Ca with HCTZ group in experiment 2. To quantitate the expression of TRPV5, calbindin-D(28K), and sodium transporters, western blotting was performed. In both experiments, HCTZ significantly decreased urinary calcium excretion. TRPV5 protein abundance decreased in all hypercalciuric rats, and restored by HCTZ in both high salt with HCTZ and high Ca with HCTZ group. Calbindin-D(28K) protein abundance increased in the high salt and high salt with HCTZ groups, but did not differ among groups in experiment 2. Protein abundance of NHE3 and NKCC2 decreased in all hypercalciuric rats, and were restored by HCTZ in only high Ca-induced hypercalciuric rats. In summary, protein abundance of TRPV5, NHE3, and NKCC2 decreased in all hypercalciuric rats. The hypocalciuric effect of HCTZ is associated with increased protein abundance of TRPV5 in high salt or calcium diet-induced hypercalciuric rats. |Animals[MESH] |Biological Transport[MESH] |Calbindin 1[MESH] |Calbindins[MESH] |Calcium Channels/chemistry[MESH] |Calcium/urine[MESH] |Hydrochlorothiazide/pharmacology[MESH] |Hypercalciuria/*therapy[MESH] |Male[MESH] |Models, Biological[MESH] |Rats[MESH] |Rats, Sprague-Dawley[MESH] |S100 Calcium Binding Protein G/*biosynthesis[MESH] |Sodium-Hydrogen Exchanger 3[MESH] |Sodium-Hydrogen Exchangers/chemistry[MESH] |Sodium-Potassium-Chloride Symporters/metabolism[MESH] |Sodium/*metabolism[MESH] |Solute Carrier Family 12, Member 1[MESH] |TRPV Cation Channels/*biosynthesis/chemistry[MESH]Effects of thiazide on the expression of TRPV5, calbindin-D28K, and so(epmc).pdf DeepDyve Pubget Overpricing