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10.1007/s00467-008-1021-9 http://scihub22266oqcxt.onion/10.1007/s00467-008-1021-9 18958502!2716751!18958502     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Pediatr+Nephrol 2009 ; 24 (6): 1113-20 Nephropedia Template TP {{tp|p=18958502|t=2009. Renin-angiotensin system-growth factor cross-talk: a novel mechanism for ureteric bud morphogenesis |pdf=|usr=}}{{18958502}} gab.com Text Renin-angiotensin system-growth factor cross-talk: a novel mechanism for ureteric bud morphogenesis JO: Pediatr Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=18958502 #FOAvip The renin-angiotensin system (RAS) plays a critical role in kidney development. Mutations in the genes encoding components of the RAS cause a spectrum of congenital abnormalities of the kidney and renal collecting system, ranging from hypoplasia of the renal medulla and hydronephrosis in mice to renal tubular dysgenesis in humans. However, the mechanisms by which an intact RAS controls proper renal system development and how aberrations in the RAS result in abnormal kidney and renal collecting system development are poorly understood. The renal collecting system originates from the ureteric bud (UB). A number of transcription and growth factors regulate UB branching morphogenesis to ultimately form the ureter, pelvis, calyces, medullary, and cortical collecting ducts. Importantly, UB morphogenesis is a key developmental process that controls organogenesis of the entire metanephros. This review emphasizes emerging insights into the role for the RAS in UB morphogenesis and explores the mechanisms whereby RAS regulates this important process. A conceptual framework derived from recent work indicates that cooperation between the angiotensin II AT(1) receptor and receptor tyrosine kinase signaling performs essential functions during renal collecting system development via control of UB branching morphogenesis. Twit Text FOAVip Renin-angiotensin system-growth factor cross-talk: a novel mechanism for ureteric bud morphogenesis ????Pediatr Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=18958502 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=18958502 #FOAvip English Wikipedia <ref>{{Cite pmid|18958502}}</ref> Renin-angiotensin system-growth factor cross-talk: a novel mechanism for ureteric bud morphogenesis #MMPMID18958502 Yosypiv IV Pediatr Nephrol 2009[Jun]; 24 (6): 1113-20 PMID18958502show ga The renin-angiotensin system (RAS) plays a critical role in kidney development. Mutations in the genes encoding components of the RAS cause a spectrum of congenital abnormalities of the kidney and renal collecting system, ranging from hypoplasia of the renal medulla and hydronephrosis in mice to renal tubular dysgenesis in humans. However, the mechanisms by which an intact RAS controls proper renal system development and how aberrations in the RAS result in abnormal kidney and renal collecting system development are poorly understood. The renal collecting system originates from the ureteric bud (UB). A number of transcription and growth factors regulate UB branching morphogenesis to ultimately form the ureter, pelvis, calyces, medullary, and cortical collecting ducts. Importantly, UB morphogenesis is a key developmental process that controls organogenesis of the entire metanephros. This review emphasizes emerging insights into the role for the RAS in UB morphogenesis and explores the mechanisms whereby RAS regulates this important process. A conceptual framework derived from recent work indicates that cooperation between the angiotensin II AT(1) receptor and receptor tyrosine kinase signaling performs essential functions during renal collecting system development via control of UB branching morphogenesis. |Animals[MESH] |Embryonic Development/genetics[MESH] |ErbB Receptors/metabolism[MESH] |Gene Expression Regulation, Developmental[MESH] |Glial Cell Line-Derived Neurotrophic Factor/metabolism[MESH] |Humans[MESH] |Kidney/metabolism[MESH] |Morphogenesis/*genetics[MESH] |Proto-Oncogene Proteins c-ret/metabolism[MESH] |Renin-Angiotensin System/*physiology[MESH]Renin-angiotensin system-growth factor cross-talk: a novel mechanism f(epmc).pdf DeepDyve Pubget Overpricing