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10.1007/s00424-008-0593-3

http://scihub22266oqcxt.onion/10.1007/s00424-008-0593-3
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18839206!2730119!18839206
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suck abstract from ncbi


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pmid18839206      Pflugers+Arch 2009 ; 458 (1): 157-68
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  • Regulation of potassium (K) handling in the renal collecting duct #MMPMID18839206
  • Wang WH; Giebisch G
  • Pflugers Arch 2009[May]; 458 (1): 157-68 PMID18839206show ga
  • This review provides an overview of the molecular mechanisms of K transport in the mammalian connecting tubule (CNT) and cortical collecting duct (CCD), both nephron segments responsible for the regulation of renal K secretion. Aldosterone and dietary K intake are two of the most important factors regulating K secretion in the CNT and CCD. Recently, angiotensin II (AngII) has also been shown to play a role in the regulation of K secretion. In addition, genetic and molecular biological approaches have further identified new mechanisms by which aldosterone and dietary K intake regulate K transport. Thus, the interaction between serum-glucocorticoid-induced kinase 1 (SGK1) and with-no-lysine kinase 4 (WNK4) plays a significant role in mediating the effect of aldosterone on ROMK (Kir1.1), an important apical K channel modulating K secretion. Recent evidence suggests that WNK1, mitogen-activated protein kinases such as P38, ERK, and Src family protein tyrosine kinase are involved in mediating the effect of low K intake on apical K secretory channels.
  • |Aldosterone/physiology[MESH]
  • |Angiotensin II/physiology[MESH]
  • |Animals[MESH]
  • |Extracellular Signal-Regulated MAP Kinases/physiology[MESH]
  • |Gastrointestinal Hormones/physiology[MESH]
  • |Immediate-Early Proteins/metabolism[MESH]
  • |Kidney Tubules, Collecting/drug effects/*metabolism[MESH]
  • |Natriuretic Peptides/physiology[MESH]
  • |Potassium Channels, Inwardly Rectifying/physiology[MESH]
  • |Potassium, Dietary/*pharmacology[MESH]
  • |Potassium/*metabolism[MESH]
  • |Protein Serine-Threonine Kinases/metabolism/physiology[MESH]
  • |p38 Mitogen-Activated Protein Kinases/physiology[MESH]


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