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10.1159/000163651 http://scihub22266oqcxt.onion/10.1159/000163651 1883518!ä!1883518 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Pathobiology 1991 ; 59 (4): 227-31 Nephropedia Template TP {{tp|p=1883518|t=1991. Influenza A virus infects macrophages and stimulates release of tumor necrosis factor-alpha |pdf=|usr=}}{{1883518}} gab.com Text Influenza A virus infects macrophages and stimulates release of tumor necrosis factor-alpha JO: Pathobiology http://www.kidney.de/pget.php?&tw=1&pmid=1883518 #FOAvip The clinical picture of influenza A virus infections indicates that release of tumor necrosis factor-alpha (TNF-alpha) may be involved. In the present study we exposed the murine macrophage line PU5-1.8 to influenza A virus and observed a productive infection which was followed by subsequent cell death. Infection of macrophages was accompanied by TNF-alpha mRNA accumulation and TNF-alpha release. TNF-alpha production could only be induced by live virus whereas interferon release was also stimulated by inactivated virus. When virus-infected macrophages were exposed to low amounts of lipopolysaccharide (LPS; 1-10 ng/ml) TNF-alpha production was strongly potentiated. These data show that low LPS concentrations could readily trigger a high TNF-alpha release from influenza-A-virus-infected macrophages which could, at least partially, explain the serious complications of combined influenza A virus and bacterial infections. Twit Text FOAVip Influenza A virus infects macrophages and stimulates release of tumor necrosis factor-alpha ????Pathobiology http://www.kidney.de/pget.php?&tw=1&pmid=1883518 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=1883518 #FOAvip English Wikipedia <ref>{{Cite pmid|1883518}}</ref> Influenza A virus infects macrophages and stimulates release of tumor necrosis factor-alpha #MMPMID1883518 Hinder F; Schmidt A; Gong JH; Bender A; Sprenger H; Nain M; Gemsa D Pathobiology 1991[]; 59 (4): 227-31 PMID1883518show ga The clinical picture of influenza A virus infections indicates that release of tumor necrosis factor-alpha (TNF-alpha) may be involved. In the present study we exposed the murine macrophage line PU5-1.8 to influenza A virus and observed a productive infection which was followed by subsequent cell death. Infection of macrophages was accompanied by TNF-alpha mRNA accumulation and TNF-alpha release. TNF-alpha production could only be induced by live virus whereas interferon release was also stimulated by inactivated virus. When virus-infected macrophages were exposed to low amounts of lipopolysaccharide (LPS; 1-10 ng/ml) TNF-alpha production was strongly potentiated. These data show that low LPS concentrations could readily trigger a high TNF-alpha release from influenza-A-virus-infected macrophages which could, at least partially, explain the serious complications of combined influenza A virus and bacterial infections. |Animals[MESH] |Cell Line[MESH] |Influenza A virus/*pathogenicity[MESH] |Kinetics[MESH] |Lipopolysaccharides/pharmacology[MESH] |Macrophages/metabolism/*microbiology[MESH] |Orthomyxoviridae Infections/physiopathology[MESH] |RNA, Messenger/metabolism[MESH]Influenza A virus infects macrophages and stimulates release of tumor (epmc).pdf DeepDyve Pubget Overpricing