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http://scihub22266oqcxt.onion/ 18788589!ä!18788589 Warning: Undefined variable $yww in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 538 Warning: Undefined variable $yww in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 538       Zhonghua+Bing+Li+Xue+Za+Zhi 2008 ; 37 (3): 150-4 Nephropedia Template TP {{tp|p=18788589|t=2008. Histologic and ultrastructural studies of the patient died of highly pathogenic H5N1 avian influenza virus infection in China |pdf=|usr=}}{{18788589}} gab.com Text Histologic and ultrastructural studies of the patient died of highly pathogenic H5N1 avian influenza virus infection in China JO: Zhonghua Bing Li Xue Za Zhi http://www.kidney.de/pget.php?&tw=1&pmid=18788589 #FOAvip OBJECTIVE: To explore histopathologic and ultrastructural characteristics of human avian influenza (AI) infection and related etiological pathogenesis. METHODS: Postmortem lung and heart samples were collected from the patient who died of avian influenza virus infection on November 29, 2003 in China. Light and electron microscopy, immunohistochemistry and histochemistry were used to investigate the pathological changes. RESULTS: The main pathological findings included extensive pulmonary consolidation, hemorrhage, pulmonary edema and local hemorrhagic infarct. The lamina of alveoli and bronchioles were abundantly filled with protein-rich fluid, erythrocytes, fibrin and cell debris admixed with many neutrophilis, macrophages, lymphocytes and a few of monokaryon and multinuclear giant cells. Hyaline membranes were formed. Local pulmonary tissues were heavily damaged by hemorrhage and necrosis. Alveolar septum was disintegrated. Mesenchymal edema with a few of macrophages infiltration of heart was found. Electron microscopy showed the avian influenza A virus-like particles (type C and type A) of 80 - 120 nm diameter and envelopes in the cytoplasm of pneumocytes and endothelial cells. CONCLUSIONS: Fatal pneumonia associated with highly pathogenic avian influenza A virus (H5N1) infection leads to extensive pulmonary consolidation, edema and marked hemorrhagic necrosis and inflammation. Electron microscopy can identify avian influenza A virus-like particles. The findings may offer an important theoretical basis for clinical diagnosis and treatment. Twit Text FOAVip Histologic and ultrastructural studies of the patient died of highly pathogenic H5N1 avian influenza virus infection in China ????Zhonghua Bing Li Xue Za Zhi http://www.kidney.de/pget.php?&tw=1&pmid=18788589 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=18788589 #FOAvip English Wikipedia <ref>{{Cite pmid|18788589}}</ref> Histologic and ultrastructural studies of the patient died of highly pathogenic H5N1 avian influenza virus infection in China #MMPMID18788589 Li N; Zhu QY; Yu Q; Wang W; Wang YP Zhonghua Bing Li Xue Za Zhi 2008[Mar]; 37 (3): 150-4 PMID18788589show ga OBJECTIVE: To explore histopathologic and ultrastructural characteristics of human avian influenza (AI) infection and related etiological pathogenesis. METHODS: Postmortem lung and heart samples were collected from the patient who died of avian influenza virus infection on November 29, 2003 in China. Light and electron microscopy, immunohistochemistry and histochemistry were used to investigate the pathological changes. RESULTS: The main pathological findings included extensive pulmonary consolidation, hemorrhage, pulmonary edema and local hemorrhagic infarct. The lamina of alveoli and bronchioles were abundantly filled with protein-rich fluid, erythrocytes, fibrin and cell debris admixed with many neutrophilis, macrophages, lymphocytes and a few of monokaryon and multinuclear giant cells. Hyaline membranes were formed. Local pulmonary tissues were heavily damaged by hemorrhage and necrosis. Alveolar septum was disintegrated. Mesenchymal edema with a few of macrophages infiltration of heart was found. Electron microscopy showed the avian influenza A virus-like particles (type C and type A) of 80 - 120 nm diameter and envelopes in the cytoplasm of pneumocytes and endothelial cells. CONCLUSIONS: Fatal pneumonia associated with highly pathogenic avian influenza A virus (H5N1) infection leads to extensive pulmonary consolidation, edema and marked hemorrhagic necrosis and inflammation. Electron microscopy can identify avian influenza A virus-like particles. The findings may offer an important theoretical basis for clinical diagnosis and treatment. |Animals[MESH] |Autopsy/methods[MESH] |Birds[MESH] |China[MESH] |Humans[MESH] |Influenza A Virus, H5N1 Subtype/*pathogenicity/ultrastructure[MESH] |Influenza A virus/classification[MESH] |Influenza in Birds/*pathology[MESH] |Influenza, Human/*diagnostic imaging/*pathology/surgery/virology[MESH] |Microscopy, Electron[MESH] |Ultrasonography[MESH] Histologic and ultrastructural studies of the patient died of highly (epmc).pdf DeepDyve Pubget Overpricing