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10.1016/j.semnephrol.2008.03.009 http://scihub22266oqcxt.onion/10.1016/j.semnephrol.2008.03.009 18519089!2587374!18519089     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Semin+Nephrol 2008 ; 28 (3): 289-96 Nephropedia Template TP {{tp|p=18519089|t=2008. Vasopressin and aquaporin 2 in clinical disorders of water homeostasis |pdf=|usr=}}{{18519089}} gab.com Text Vasopressin and aquaporin 2 in clinical disorders of water homeostasis JO: Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=18519089 #FOAvip Impaired urinary dilution leading to water retention and hyponatremia may occur in patients with cardiac failure, cirrhosis, pregnancy, oxytocin administration, hypothyroidism, glucocorticoid, and mineralocorticoid deficiency. The mechanisms for these defects predominantly involve the nonosmotic stimulation of arginine vasopressin release with up-regulation of aquaporin 2 water channel expression and trafficking to the apical membrane of the principal cells of the collecting duct. These perturbations are reversed by V2 vasopressin receptor antagonists. In contrast, urinary concentration defects leading to polyuria are vasopressin resistant. They may involve several factors, such as impaired countercurrent concentration secondary to down-regulation of Na-K-2Cl cotransporter. Vasopressin-resistant down-regulation of aquaporin 2 expression has also been described as a factor in impaired urinary concentration. Twit Text FOAVip Vasopressin and aquaporin 2 in clinical disorders of water homeostasis ????Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=18519089 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=18519089 #FOAvip English Wikipedia <ref>{{Cite pmid|18519089}}</ref> Vasopressin and aquaporin 2 in clinical disorders of water homeostasis #MMPMID18519089 Schrier RW Semin Nephrol 2008[May]; 28 (3): 289-96 PMID18519089show ga Impaired urinary dilution leading to water retention and hyponatremia may occur in patients with cardiac failure, cirrhosis, pregnancy, oxytocin administration, hypothyroidism, glucocorticoid, and mineralocorticoid deficiency. The mechanisms for these defects predominantly involve the nonosmotic stimulation of arginine vasopressin release with up-regulation of aquaporin 2 water channel expression and trafficking to the apical membrane of the principal cells of the collecting duct. These perturbations are reversed by V2 vasopressin receptor antagonists. In contrast, urinary concentration defects leading to polyuria are vasopressin resistant. They may involve several factors, such as impaired countercurrent concentration secondary to down-regulation of Na-K-2Cl cotransporter. Vasopressin-resistant down-regulation of aquaporin 2 expression has also been described as a factor in impaired urinary concentration. |*Homeostasis[MESH] |Aquaporin 2/*physiology[MESH] |Body Water[MESH] |Humans[MESH] |Hyponatremia/complications/*etiology[MESH] |Polyuria/complications/*etiology[MESH]Vasopressin and aquaporin 2 in clinical disorders of water homeostasis(epmc).pdf DeepDyve Pubget Overpricing