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10.1016/j.amjcard.2008.02.074 http://scihub22266oqcxt.onion/10.1016/j.amjcard.2008.02.074 18514619!ä!18514619 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Cardiol 2008 ; 101 (11A): 11E-15E Nephropedia Template TP {{tp|p=18514619|t=2008. Hypercatabolic syndrome: molecular basis and effects of nutritional supplements with amino acids |pdf=|usr=}}{{18514619}} gab.com Text Hypercatabolic syndrome: molecular basis and effects of nutritional supplements with amino acids JO: Am J Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=18514619 #FOAvip Hypercatabolic syndrome (HS) is a biochemical state characterized by increased circulating catabolic hormones (eg, cortisol, catecholamines) and inflammatory cytokines (eg, tumor necrosis factors, interleukin-1beta), and decreased anabolic insulin effects with consequent insulin resistance. The most important metabolic consequence of HS is the skeletal and cardiac muscle protein breakdown that releases amino acids (AAs), which in turn supports indispensable body energy requirements but also reduces skeletal and cardiac physiologic and metabolic functions. HS occurs in many diseases such as diabetes mellitus, chronic heart failure, chronic obstructive pulmonary disease, renal and liver failure, trauma, sepsis, and senescence. All of these conditions have predominant catabolic molecules with significant muscular wasting and metabolic impairment. Macronutrients such as AA supplements, taken together with conventional therapy, may maintain muscular protein metabolism and cell functions. Twit Text FOAVip Hypercatabolic syndrome: molecular basis and effects of nutritional supplements with amino acids ????Am J Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=18514619 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=18514619 #FOAvip English Wikipedia <ref>{{Cite pmid|18514619}}</ref> Hypercatabolic syndrome: molecular basis and effects of nutritional supplements with amino acids #MMPMID18514619 Pasini E; Aquilani R; Dioguardi FS; D'Antona G; Gheorghiade M; Taegtmeyer H Am J Cardiol 2008[Jun]; 101 (11A): 11E-15E PMID18514619show ga Hypercatabolic syndrome (HS) is a biochemical state characterized by increased circulating catabolic hormones (eg, cortisol, catecholamines) and inflammatory cytokines (eg, tumor necrosis factors, interleukin-1beta), and decreased anabolic insulin effects with consequent insulin resistance. The most important metabolic consequence of HS is the skeletal and cardiac muscle protein breakdown that releases amino acids (AAs), which in turn supports indispensable body energy requirements but also reduces skeletal and cardiac physiologic and metabolic functions. HS occurs in many diseases such as diabetes mellitus, chronic heart failure, chronic obstructive pulmonary disease, renal and liver failure, trauma, sepsis, and senescence. All of these conditions have predominant catabolic molecules with significant muscular wasting and metabolic impairment. Macronutrients such as AA supplements, taken together with conventional therapy, may maintain muscular protein metabolism and cell functions. |*Dietary Supplements[MESH] |Administration, Oral[MESH] |Amino Acids/administration & dosage/*therapeutic use[MESH] |Cachexia/metabolism/*therapy[MESH] |Chronic Disease[MESH] |Energy Metabolism/physiology[MESH] |Gluconeogenesis/physiology[MESH] |Heart Failure/metabolism[MESH] |Humans[MESH] |Insulin Resistance/physiology[MESH] |Muscle, Skeletal/metabolism[MESH] |Myocytes, Cardiac/metabolism[MESH]Hypercatabolic syndrome: molecular basis and effects of nutritional su(epmc).pdf DeepDyve Pubget Overpricing