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10.1111/j.1365-2249.2006.03286.x http://scihub22266oqcxt.onion/10.1111/j.1365-2249.2006.03286.x 17302890!1810484!17302890     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Exp+Immunol 2007 ; 147 (3): 419-25 Nephropedia Template TP {{tp|p=17302890|t=2007. Cytokine responses in acute and persistent human parvovirus B19 infection |pdf=|usr=}}{{17302890}} gab.com Text Cytokine responses in acute and persistent human parvovirus B19 infection JO: Clin Exp Immunol http://www.kidney.de/pget.php?&tw=1&pmid=17302890 #FOAvip The aim of this study was to characterize the proinflammatory and T helper (Th)1/Th2 cytokine responses during acute parvovirus B19 (B19) infection and determine whether an imbalance of the Th1/Th2 cytokine pattern is related to persistent B19 infection. Cytokines were quantified by multiplex beads immunoassay in serum from B19-infected patients and controls. The cytokine responses were correlated with B19 serology, quantitative B19 DNA levels and clinical symptoms. In addition to a proinflammatory response, elevated levels of the Th1 type of cytokines interleukin (IL)-2, IL-12 and IL-15 were evident at time of the initial peak of B19 viral load in a few patients during acute infection. This pattern was seen in the absence of an interferon (IFN)-gamma response. During follow-up (20-130 weeks post-acute infection) some of these patients had a sustained Th1 cytokine response. The Th1 cytokine response correlated with the previously identified sustained CD8+ T cell response and viraemia. A cross-sectional study on patients with persistent B19 infection showed no apparent imbalance of their cytokine pattern, except for an elevated level of IFN-gamma response. No general immunodeficiency was diagnosed as an explanation for the viral persistence in this later group. Neither the acutely infected nor the persistently infected patients demonstrated a Th2 cytokine response. In conclusion, the acutely infected patients demonstrated a sustained Th1 cytokine response whereas the persistently infected patients did not exhibit an apparent imbalance of their cytokine pattern except for an elevated IFN-gamma response. Twit Text FOAVip Cytokine responses in acute and persistent human parvovirus B19 infection ????Clin Exp Immunol http://www.kidney.de/pget.php?&tw=1&pmid=17302890 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=17302890 #FOAvip English Wikipedia <ref>{{Cite pmid|17302890}}</ref> Cytokine responses in acute and persistent human parvovirus B19 infection #MMPMID17302890 Isa A; Lundqvist A; Lindblom A; Tolfvenstam T; Broliden K Clin Exp Immunol 2007[Mar]; 147 (3): 419-25 PMID17302890show ga The aim of this study was to characterize the proinflammatory and T helper (Th)1/Th2 cytokine responses during acute parvovirus B19 (B19) infection and determine whether an imbalance of the Th1/Th2 cytokine pattern is related to persistent B19 infection. Cytokines were quantified by multiplex beads immunoassay in serum from B19-infected patients and controls. The cytokine responses were correlated with B19 serology, quantitative B19 DNA levels and clinical symptoms. In addition to a proinflammatory response, elevated levels of the Th1 type of cytokines interleukin (IL)-2, IL-12 and IL-15 were evident at time of the initial peak of B19 viral load in a few patients during acute infection. This pattern was seen in the absence of an interferon (IFN)-gamma response. During follow-up (20-130 weeks post-acute infection) some of these patients had a sustained Th1 cytokine response. The Th1 cytokine response correlated with the previously identified sustained CD8+ T cell response and viraemia. A cross-sectional study on patients with persistent B19 infection showed no apparent imbalance of their cytokine pattern, except for an elevated level of IFN-gamma response. No general immunodeficiency was diagnosed as an explanation for the viral persistence in this later group. Neither the acutely infected nor the persistently infected patients demonstrated a Th2 cytokine response. In conclusion, the acutely infected patients demonstrated a sustained Th1 cytokine response whereas the persistently infected patients did not exhibit an apparent imbalance of their cytokine pattern except for an elevated IFN-gamma response. |Acute Disease[MESH] |Adult[MESH] |Chronic Disease[MESH] |Cross-Sectional Studies[MESH] |Cytokines/*blood[MESH] |DNA, Viral/blood[MESH] |Female[MESH] |Histocompatibility Testing[MESH] |Humans[MESH] |Inflammation Mediators/blood[MESH] |Interferon-gamma/blood[MESH] |Middle Aged[MESH] |Parvoviridae Infections/*immunology/virology[MESH] |Parvovirus B19, Human/*isolation & purification[MESH] |Th1 Cells/immunology[MESH] |Th2 Cells/immunology[MESH]Cytokine responses in acute and persistent human parvovirus B19 infect(epmc).pdf DeepDyve Pubget Overpricing