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10.1016/j.semcdb.2006.11.003 http://scihub22266oqcxt.onion/10.1016/j.semcdb.2006.11.003 17116414!2705932!17116414     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Semin+Cell+Dev+Biol 2006 ; 17 (6): 667-74 Nephropedia Template TP {{tp|p=17116414|t=2006. TRPC6 in glomerular health and disease: what we know and what we believe |pdf=|usr=}}{{17116414}} gab.com Text TRPC6 in glomerular health and disease: what we know and what we believe JO: Semin Cell Dev Biol http://www.kidney.de/pget.php?&tw=1&pmid=17116414 #FOAvip Mutations in TRPC6, a member of the transient receptor potential (TRP) superfamily of non-selective cation channels, have been identified as causing a familial form of focal segmental glomerulosclerosis, a disease characterized by proteinuria and progressive renal failure. Here we review the effect of disease-associated mutations on TRPC6 function and place TRPC6 within the context of other proteins central to glomerular and podocyte function. Finally, the known roles of TRPC6 in the kidney and other organ systems are used as a framework to discuss possible signaling pathways that TRPC6 may modulate during normal glomerular function and in disease states. Twit Text FOAVip TRPC6 in glomerular health and disease: what we know and what we believe ????Semin Cell Dev Biol http://www.kidney.de/pget.php?&tw=1&pmid=17116414 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=17116414 #FOAvip English Wikipedia <ref>{{Cite pmid|17116414}}</ref> TRPC6 in glomerular health and disease: what we know and what we believe #MMPMID17116414 Schlondorff JS; Pollak MR Semin Cell Dev Biol 2006[Dec]; 17 (6): 667-74 PMID17116414show ga Mutations in TRPC6, a member of the transient receptor potential (TRP) superfamily of non-selective cation channels, have been identified as causing a familial form of focal segmental glomerulosclerosis, a disease characterized by proteinuria and progressive renal failure. Here we review the effect of disease-associated mutations on TRPC6 function and place TRPC6 within the context of other proteins central to glomerular and podocyte function. Finally, the known roles of TRPC6 in the kidney and other organ systems are used as a framework to discuss possible signaling pathways that TRPC6 may modulate during normal glomerular function and in disease states. |Animals[MESH] |Humans[MESH] |Kidney Diseases/*metabolism/*physiopathology[MESH] |Kidney Glomerulus/*metabolism/physiology/*physiopathology[MESH] |TRPC Cation Channels/*physiology[MESH]TRPC6 in glomerular health and disease: what we know and what we belie(epmc).pdf DeepDyve Pubget Overpricing